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Associations between genetic obesity susceptibility and early postnatal fat and lean mass: an individual participant meta-analysis

Associations between genetic obesity susceptibility and early postnatal fat and lean mass: an individual participant meta-analysis
Associations between genetic obesity susceptibility and early postnatal fat and lean mass: an individual participant meta-analysis
Importance

Patterns of body size and body composition associated with genetic obesity susceptibility inform the mechanisms that increase obesity risk.

Objective

To test associations between genetic obesity susceptibility, represented by a combined obesity risk-allele score, and body size or body composition at birth to age 5 years.

Design, Setting, and Participants

A total of 3031 children from 4 birth cohort studies in England, France, and Spain were included in a meta-analysis.

Exposures

A combined obesity risk-allele score was calculated from genotypes at 16 variants identified by genome-wide association studies of adult body mass index (BMI).

Main Outcomes and Measures

Outcomes were age- and sex-adjusted SD scores (SDS) for weight, length/height, BMI, fat mass, lean mass, and percentage of body fat at birth as well as at ages 1, 2 to 3, and 4 to 5 years.

Results

The obesity risk-allele score was not associated with infant size at birth; at age 1 year it was positively associated with weight (? [SE], 0.020 [0.008] SDS per allele; P?=?.009) and length (? [SE], 0.020 [0.008] SDS per allele; P?=?.01), but not with BMI (? [SE], 0.013 [0.008] SDS per allele; P?=?.11). At age 2 to 3 years these associations were stronger (weight: ? [SE], 0.033 [0.008] SDS per allele; P?<?.001; height: ? [SE], 0.025 [0.008] SDS per allele; P?<?.001) and were also seen for BMI (? [SE], 0.024 [0.008] SDS per allele; P?=?.003). The obesity risk-allele score was positively associated with both postnatal fat mass (1 year: ? [SE], 0.032?[0.017] SDS per allele; P?=?.05; 2-3 years: ? [SE], 0.049?[0.018] SDS per allele; P?=?.006; and 4-5 years: ? [SE], 0.028?[0.011] SDS per allele; P?=?.009) and postnatal lean mass (1 year: ? [SE], 0.038 [0.014] SDS per allele; P?=?.008; 2-3 years: ? [SE], 0.064 [0.017] SDS per allele; P?<?.001; and 4-5 years: ? [SE], 0.047 [0.011] SDS per allele; P?<?.001), but not with the percentage of body fat (P?>?.15 at all ages).

Conclusions and Relevance

Genetic obesity susceptibility appears to promote a normally partitioned increase in early postnatal, but not prenatal, growth. These findings suggest that symmetrical rapid growth may identify infants with high life-long susceptibility for obesity.
0098-7484
1122-1130
Elks, C.E.
9baecf7a-3a29-4c66-a3ff-29ef504a3b67
Heude, B.
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de Zegher, F.
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Barton, S.J.
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Clement, K.
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Inskip, H.M.
5fb4470a-9379-49b2-a533-9da8e61058b7
Koudou, Y.
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Cooper, C.
e05f5612-b493-4273-9b71-9e0ce32bdad6
Dunger, D.B.
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Ibanez, L.
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Charles, M.A.
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Ong, K.K.
c7a86c7d-0b6e-4b35-bcc9-59895067c82e
Elks, C.E.
9baecf7a-3a29-4c66-a3ff-29ef504a3b67
Heude, B.
c8751484-c906-418f-9b23-45530c82fc65
de Zegher, F.
84af77aa-2ab1-4fc6-9f19-62a7cc0fe51b
Barton, S.J.
4f674382-ca0b-44ad-9670-e71a0b134ef0
Clement, K.
1b2b0890-ca34-4f7f-a8f7-528b02175e32
Inskip, H.M.
5fb4470a-9379-49b2-a533-9da8e61058b7
Koudou, Y.
04a5fe54-9e2a-4db8-b691-f9b3899b08bd
Cooper, C.
e05f5612-b493-4273-9b71-9e0ce32bdad6
Dunger, D.B.
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Ibanez, L.
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Charles, M.A.
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Ong, K.K.
c7a86c7d-0b6e-4b35-bcc9-59895067c82e

Elks, C.E., Heude, B., de Zegher, F., Barton, S.J., Clement, K., Inskip, H.M., Koudou, Y., Cooper, C., Dunger, D.B., Ibanez, L., Charles, M.A. and Ong, K.K. (2014) Associations between genetic obesity susceptibility and early postnatal fat and lean mass: an individual participant meta-analysis. JAMA, 168 (12), 1122-1130. (doi:10.1001/jamapediatrics.2014.1619). (PMID:25329327)

Record type: Article

Abstract

Importance

Patterns of body size and body composition associated with genetic obesity susceptibility inform the mechanisms that increase obesity risk.

Objective

To test associations between genetic obesity susceptibility, represented by a combined obesity risk-allele score, and body size or body composition at birth to age 5 years.

Design, Setting, and Participants

A total of 3031 children from 4 birth cohort studies in England, France, and Spain were included in a meta-analysis.

Exposures

A combined obesity risk-allele score was calculated from genotypes at 16 variants identified by genome-wide association studies of adult body mass index (BMI).

Main Outcomes and Measures

Outcomes were age- and sex-adjusted SD scores (SDS) for weight, length/height, BMI, fat mass, lean mass, and percentage of body fat at birth as well as at ages 1, 2 to 3, and 4 to 5 years.

Results

The obesity risk-allele score was not associated with infant size at birth; at age 1 year it was positively associated with weight (? [SE], 0.020 [0.008] SDS per allele; P?=?.009) and length (? [SE], 0.020 [0.008] SDS per allele; P?=?.01), but not with BMI (? [SE], 0.013 [0.008] SDS per allele; P?=?.11). At age 2 to 3 years these associations were stronger (weight: ? [SE], 0.033 [0.008] SDS per allele; P?<?.001; height: ? [SE], 0.025 [0.008] SDS per allele; P?<?.001) and were also seen for BMI (? [SE], 0.024 [0.008] SDS per allele; P?=?.003). The obesity risk-allele score was positively associated with both postnatal fat mass (1 year: ? [SE], 0.032?[0.017] SDS per allele; P?=?.05; 2-3 years: ? [SE], 0.049?[0.018] SDS per allele; P?=?.006; and 4-5 years: ? [SE], 0.028?[0.011] SDS per allele; P?=?.009) and postnatal lean mass (1 year: ? [SE], 0.038 [0.014] SDS per allele; P?=?.008; 2-3 years: ? [SE], 0.064 [0.017] SDS per allele; P?<?.001; and 4-5 years: ? [SE], 0.047 [0.011] SDS per allele; P?<?.001), but not with the percentage of body fat (P?>?.15 at all ages).

Conclusions and Relevance

Genetic obesity susceptibility appears to promote a normally partitioned increase in early postnatal, but not prenatal, growth. These findings suggest that symmetrical rapid growth may identify infants with high life-long susceptibility for obesity.

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More information

Published date: December 2014
Organisations: Human Development & Health

Identifiers

Local EPrints ID: 373546
URI: http://eprints.soton.ac.uk/id/eprint/373546
ISSN: 0098-7484
PURE UUID: d1644ecd-3eaf-4a2b-b652-c1dbe8426017
ORCID for S.J. Barton: ORCID iD orcid.org/0000-0003-4963-4242
ORCID for H.M. Inskip: ORCID iD orcid.org/0000-0001-8897-1749
ORCID for C. Cooper: ORCID iD orcid.org/0000-0003-3510-0709

Catalogue record

Date deposited: 21 Jan 2015 12:40
Last modified: 18 Mar 2024 02:54

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Contributors

Author: C.E. Elks
Author: B. Heude
Author: F. de Zegher
Author: S.J. Barton ORCID iD
Author: K. Clement
Author: H.M. Inskip ORCID iD
Author: Y. Koudou
Author: C. Cooper ORCID iD
Author: D.B. Dunger
Author: L. Ibanez
Author: M.A. Charles
Author: K.K. Ong

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