The University of Southampton
University of Southampton Institutional Repository

Human follicular lymphoma cells contain oligomannose glycans in the antigen-binding site of the B-cell receptor

Human follicular lymphoma cells contain oligomannose glycans in the antigen-binding site of the B-cell receptor
Human follicular lymphoma cells contain oligomannose glycans in the antigen-binding site of the B-cell receptor
Expression of surface immunoglobulin appears critical for the growth and survival of B-cell lymphomas. In follicular lymphoma, we found previously that the Ig variable (V) regions in the B-cell receptor express a strikingly high incidence of N-glycosylation sequons, NX(S/T). These potential glycosylation sites are introduced by somatic mutation and are lymphoma-specific, pointing to their involvement in tumor pathogenesis. Analysis of the V region sugars from lymphoma-derived IgG/IgM reveals that they are mostly oligomannose and, remarkably, are located in the antigen-binding site, possibly precluding conventional antigen binding. The Fc region contains complex glycans, confirming that the normal glycan processing pathway is intact. Binding studies indicate that the oligomannose glycans occupying the V regions are accessible to mannose-binding lectin. These findings suggest a potential contribution to lymphoma pathogenesis involving antigen-independent interaction of surface immunoglobulin of the B-cell receptor with mannose-binding molecules of innate immunity in the germinal center.
variable region, indicate, surface, pathogenesis, receptor, surface-immunoglobulin, molecules, survival, c-type lectins, follicular lymphoma, dendritic cells, antigen, time, b-cell receptor, mutation, expression, region, involvement, lymphoma, burkitts-lymphoma, pathway, tumor, b-cell lymphoma, rheumatoid-arthritis, growth, fab fragment, immune-system, glycosylation sites, somatic mutation, binding, n-linked oligosaccharides, immunity, cells, innate, cell, immunoglobulin, b cell, b-cell-receptor, sites
0021-9258
7405-7415
Radcliffe, Catherine M
bc36425b-233b-4262-8b54-2dc0defc1cc6
Arnold, James N
6b2c2696-9b28-440d-b5eb-6fb65d428362
Suter, David M
71c4ff17-962d-4b6b-a78b-19da42028241
Wormald, Mark R
62914dc3-37e2-460c-ab7c-01712d7e09d5
Harvey, David J
8bb24417-3852-4b1f-827b-0d5d2c176744
Royle, Louise
310a52f9-a086-482c-9963-857a3af2363b
Mimura, Yusuke
f4cdf262-a937-4ba5-83d5-fac5a7d2f327
Kimura, Yoshinobu
7eccf28a-e78d-4282-ac73-8d14e6160985
Sim, Robert B
d48628cc-2ae8-4a8c-949a-4d5c35a514ec
Inogès, Susana
e993f617-cb2e-49d4-9315-7828c2728fb9
Rodriguez-Calvillo, Mercedes
155a5e4c-5fad-49d5-971d-c1fe27afefe7
Zabalegui, Natalia
c82f0fa7-f541-4f0a-806d-4547602c0546
de Cerio, Ascensión López-Díaz
fe016e16-510a-493f-bee1-38578257129f
Potter, Kathleen N
86a99047-494b-405b-a3f7-650c1dcd5838
Mockridge, C Ian
327aef17-4837-4f2a-a93b-3d17cd1a7f9f
Dwek, Raymond A
5fb1a0ce-1ac6-4825-8ff0-f62c7164ff0c
Bendandi, Maurizio
490d18e0-a53b-4f70-baf8-ebaed7ba234f
Rudd, Pauline M
4bbd1e70-98ae-4c28-84e6-c6a18d98e7ee
Stevenson, Freda K
ba803747-c0ac-409f-a9c2-b61fde009f8c
Radcliffe, Catherine M
bc36425b-233b-4262-8b54-2dc0defc1cc6
Arnold, James N
6b2c2696-9b28-440d-b5eb-6fb65d428362
Suter, David M
71c4ff17-962d-4b6b-a78b-19da42028241
Wormald, Mark R
62914dc3-37e2-460c-ab7c-01712d7e09d5
Harvey, David J
8bb24417-3852-4b1f-827b-0d5d2c176744
Royle, Louise
310a52f9-a086-482c-9963-857a3af2363b
Mimura, Yusuke
f4cdf262-a937-4ba5-83d5-fac5a7d2f327
Kimura, Yoshinobu
7eccf28a-e78d-4282-ac73-8d14e6160985
Sim, Robert B
d48628cc-2ae8-4a8c-949a-4d5c35a514ec
Inogès, Susana
e993f617-cb2e-49d4-9315-7828c2728fb9
Rodriguez-Calvillo, Mercedes
155a5e4c-5fad-49d5-971d-c1fe27afefe7
Zabalegui, Natalia
c82f0fa7-f541-4f0a-806d-4547602c0546
de Cerio, Ascensión López-Díaz
fe016e16-510a-493f-bee1-38578257129f
Potter, Kathleen N
86a99047-494b-405b-a3f7-650c1dcd5838
Mockridge, C Ian
327aef17-4837-4f2a-a93b-3d17cd1a7f9f
Dwek, Raymond A
5fb1a0ce-1ac6-4825-8ff0-f62c7164ff0c
Bendandi, Maurizio
490d18e0-a53b-4f70-baf8-ebaed7ba234f
Rudd, Pauline M
4bbd1e70-98ae-4c28-84e6-c6a18d98e7ee
Stevenson, Freda K
ba803747-c0ac-409f-a9c2-b61fde009f8c

Radcliffe, Catherine M, Arnold, James N, Suter, David M, Wormald, Mark R, Harvey, David J, Royle, Louise, Mimura, Yusuke, Kimura, Yoshinobu, Sim, Robert B, Inogès, Susana, Rodriguez-Calvillo, Mercedes, Zabalegui, Natalia, de Cerio, Ascensión López-Díaz, Potter, Kathleen N, Mockridge, C Ian, Dwek, Raymond A, Bendandi, Maurizio, Rudd, Pauline M and Stevenson, Freda K (2007) Human follicular lymphoma cells contain oligomannose glycans in the antigen-binding site of the B-cell receptor. The Journal of Biological Chemistry, 282 (10), 7405-7415. (doi:10.1074/jbc.M602690200).

Record type: Article

Abstract

Expression of surface immunoglobulin appears critical for the growth and survival of B-cell lymphomas. In follicular lymphoma, we found previously that the Ig variable (V) regions in the B-cell receptor express a strikingly high incidence of N-glycosylation sequons, NX(S/T). These potential glycosylation sites are introduced by somatic mutation and are lymphoma-specific, pointing to their involvement in tumor pathogenesis. Analysis of the V region sugars from lymphoma-derived IgG/IgM reveals that they are mostly oligomannose and, remarkably, are located in the antigen-binding site, possibly precluding conventional antigen binding. The Fc region contains complex glycans, confirming that the normal glycan processing pathway is intact. Binding studies indicate that the oligomannose glycans occupying the V regions are accessible to mannose-binding lectin. These findings suggest a potential contribution to lymphoma pathogenesis involving antigen-independent interaction of surface immunoglobulin of the B-cell receptor with mannose-binding molecules of innate immunity in the germinal center.

This record has no associated files available for download.

More information

Published date: 9 March 2007
Keywords: variable region, indicate, surface, pathogenesis, receptor, surface-immunoglobulin, molecules, survival, c-type lectins, follicular lymphoma, dendritic cells, antigen, time, b-cell receptor, mutation, expression, region, involvement, lymphoma, burkitts-lymphoma, pathway, tumor, b-cell lymphoma, rheumatoid-arthritis, growth, fab fragment, immune-system, glycosylation sites, somatic mutation, binding, n-linked oligosaccharides, immunity, cells, innate, cell, immunoglobulin, b cell, b-cell-receptor, sites

Identifiers

Local EPrints ID: 157787
URI: http://eprints.soton.ac.uk/id/eprint/157787
ISSN: 0021-9258
PURE UUID: b84da807-3377-4421-baad-aff943466d8a
ORCID for Freda K Stevenson: ORCID iD orcid.org/0000-0002-0933-5021

Catalogue record

Date deposited: 10 Jun 2010 14:11
Last modified: 14 Mar 2024 02:40

Export record

Altmetrics

Contributors

Author: Catherine M Radcliffe
Author: James N Arnold
Author: David M Suter
Author: Mark R Wormald
Author: David J Harvey
Author: Louise Royle
Author: Yusuke Mimura
Author: Yoshinobu Kimura
Author: Robert B Sim
Author: Susana Inogès
Author: Mercedes Rodriguez-Calvillo
Author: Natalia Zabalegui
Author: Ascensión López-Díaz de Cerio
Author: C Ian Mockridge
Author: Raymond A Dwek
Author: Maurizio Bendandi
Author: Pauline M Rudd

Download statistics

Downloads from ePrints over the past year. Other digital versions may also be available to download e.g. from the publisher's website.

View more statistics

Atom RSS 1.0 RSS 2.0

Contact ePrints Soton: eprints@soton.ac.uk

ePrints Soton supports OAI 2.0 with a base URL of http://eprints.soton.ac.uk/cgi/oai2

This repository has been built using EPrints software, developed at the University of Southampton, but available to everyone to use.

We use cookies to ensure that we give you the best experience on our website. If you continue without changing your settings, we will assume that you are happy to receive cookies on the University of Southampton website.

×