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Betel-derived alkaloid up-regulates keratinocyte alphavbeta6 integrin expression and promotes oral submucous fibrosis

Betel-derived alkaloid up-regulates keratinocyte alphavbeta6 integrin expression and promotes oral submucous fibrosis
Betel-derived alkaloid up-regulates keratinocyte alphavbeta6 integrin expression and promotes oral submucous fibrosis
Oral submucous fibrosis (OSF) is a premalignant, fibrosing disorder of the mouth, pharynx, and oesophagus, with a malignant transformation rate of 7-13%. OSF is strongly associated with areca (betel) nut chewing and worldwide, over 5 million people are affected. As ?v?6 integrin is capable of promoting both tissue fibrosis and carcinoma invasion, we examined its expression in fibroepithelial hyperplasia and OSF. ?v?6 was markedly up-regulated in OSF, with high expression detected in 22 of 41 cases (p < 0.001). We investigated the functional role of ?v?6 using oral keratinocyte-derived cells genetically modified to express high ?v?6 (VB6), and also NTERT-immortalized oral keratinocytes, which express low ?v?6 (OKF6/TERT-1). VB6 cells showed significant ?v?6-dependent activation of TGF-?1, which induced transdifferentiation of oral fibroblasts into myofibroblasts and resulted in up-regulation of genes associated with tissue fibrosis. These experimental in vitro findings were confirmed using human clinical samples, where we showed that the stroma of OSF contained myofibroblasts and that TGF-?1-dependent Smad signalling was detectable both in keratinocytes and in myofibroblasts. We also found that arecoline, the major alkaloid of areca nuts, up-regulated keratinocyte ?v?6 expression. This was modulated through the M(4) muscarinic acetylcholine receptor and was suppressed by the M(4) antagonist, tropicamide. Arecoline-dependent ?v?6 up-regulation promoted keratinocyte migration and induced invasion, raising the possibility that this mechanism may support malignant transformation. Over 80% of OSF-related oral cancers examined had moderate/high ?v?6 expression. These data suggest that the pathogenesis of OSF may be epithelial-driven and involve arecoline-dependent up-regulation of ?v?6 integrin.
arecoline, betel, integrin, myofibroblast, oral submucous fibrosis, tgf-?1
1096-9896
366-377
Moutasim, Karwan A.
af7dd711-f6df-44f7-8c57-052bf15303af
Jenei, Veronika
04d82852-7d30-458a-bc0b-f00ae6c5dd52
Sapienza, Karen
8262ed3b-37b0-4109-af19-66afa6f589a4
Marsh, Daniel
eede5080-736a-4058-a4e1-a6b233651a35
Weinreb, Paul H.
ba79e7c2-caf7-426e-a9e7-10b1ccc74e15
Violette, Shelia M.
9eb0cfe3-28f0-4319-ba2e-1fd84c43bf4b
Lewis, Mark P.
a958b476-cbc0-4904-9348-57b5bfa6c162
Marshall, John F.
1134b21f-7f23-4ee6-aaf5-c7580b73c10b
Fortune, Farida
9fd3d817-1fb6-4abb-b5aa-ca96c26df159
Tilakaratne, Waninayaka M.
f180a84f-cced-4a8a-b416-9907383c5417
Hart, Ian R.
6a3728fc-5385-4dd4-a5e3-edd5168a0e8d
Thomas, Gareth J.
2ff54aa9-a766-416b-91ee-cf1c5be74106
Moutasim, Karwan A.
af7dd711-f6df-44f7-8c57-052bf15303af
Jenei, Veronika
04d82852-7d30-458a-bc0b-f00ae6c5dd52
Sapienza, Karen
8262ed3b-37b0-4109-af19-66afa6f589a4
Marsh, Daniel
eede5080-736a-4058-a4e1-a6b233651a35
Weinreb, Paul H.
ba79e7c2-caf7-426e-a9e7-10b1ccc74e15
Violette, Shelia M.
9eb0cfe3-28f0-4319-ba2e-1fd84c43bf4b
Lewis, Mark P.
a958b476-cbc0-4904-9348-57b5bfa6c162
Marshall, John F.
1134b21f-7f23-4ee6-aaf5-c7580b73c10b
Fortune, Farida
9fd3d817-1fb6-4abb-b5aa-ca96c26df159
Tilakaratne, Waninayaka M.
f180a84f-cced-4a8a-b416-9907383c5417
Hart, Ian R.
6a3728fc-5385-4dd4-a5e3-edd5168a0e8d
Thomas, Gareth J.
2ff54aa9-a766-416b-91ee-cf1c5be74106

Moutasim, Karwan A., Jenei, Veronika, Sapienza, Karen, Marsh, Daniel, Weinreb, Paul H., Violette, Shelia M., Lewis, Mark P., Marshall, John F., Fortune, Farida, Tilakaratne, Waninayaka M., Hart, Ian R. and Thomas, Gareth J. (2011) Betel-derived alkaloid up-regulates keratinocyte alphavbeta6 integrin expression and promotes oral submucous fibrosis. The Journal of Pathology, 223 (3), 366-377. (doi:10.1002/path.2786). (PMID:21171082)

Record type: Article

Abstract

Oral submucous fibrosis (OSF) is a premalignant, fibrosing disorder of the mouth, pharynx, and oesophagus, with a malignant transformation rate of 7-13%. OSF is strongly associated with areca (betel) nut chewing and worldwide, over 5 million people are affected. As ?v?6 integrin is capable of promoting both tissue fibrosis and carcinoma invasion, we examined its expression in fibroepithelial hyperplasia and OSF. ?v?6 was markedly up-regulated in OSF, with high expression detected in 22 of 41 cases (p < 0.001). We investigated the functional role of ?v?6 using oral keratinocyte-derived cells genetically modified to express high ?v?6 (VB6), and also NTERT-immortalized oral keratinocytes, which express low ?v?6 (OKF6/TERT-1). VB6 cells showed significant ?v?6-dependent activation of TGF-?1, which induced transdifferentiation of oral fibroblasts into myofibroblasts and resulted in up-regulation of genes associated with tissue fibrosis. These experimental in vitro findings were confirmed using human clinical samples, where we showed that the stroma of OSF contained myofibroblasts and that TGF-?1-dependent Smad signalling was detectable both in keratinocytes and in myofibroblasts. We also found that arecoline, the major alkaloid of areca nuts, up-regulated keratinocyte ?v?6 expression. This was modulated through the M(4) muscarinic acetylcholine receptor and was suppressed by the M(4) antagonist, tropicamide. Arecoline-dependent ?v?6 up-regulation promoted keratinocyte migration and induced invasion, raising the possibility that this mechanism may support malignant transformation. Over 80% of OSF-related oral cancers examined had moderate/high ?v?6 expression. These data suggest that the pathogenesis of OSF may be epithelial-driven and involve arecoline-dependent up-regulation of ?v?6 integrin.

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More information

Published date: February 2011
Keywords: arecoline, betel, integrin, myofibroblast, oral submucous fibrosis, tgf-?1
Organisations: Cancer Sciences

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Local EPrints ID: 182283
URI: http://eprints.soton.ac.uk/id/eprint/182283
ISSN: 1096-9896
PURE UUID: 7d34d1f6-b0b3-4734-8a7c-b7a2b6708666

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Date deposited: 26 Apr 2011 14:15
Last modified: 14 Mar 2024 02:59

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Contributors

Author: Veronika Jenei
Author: Karen Sapienza
Author: Daniel Marsh
Author: Paul H. Weinreb
Author: Shelia M. Violette
Author: Mark P. Lewis
Author: John F. Marshall
Author: Farida Fortune
Author: Waninayaka M. Tilakaratne
Author: Ian R. Hart

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