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A functional variant of the serotonin transporter gene (SLC6A4) moderates impulsive choice in attention-deficit/hyperactivity disorder boys and siblings

A functional variant of the serotonin transporter gene (SLC6A4) moderates impulsive choice in attention-deficit/hyperactivity disorder boys and siblings
A functional variant of the serotonin transporter gene (SLC6A4) moderates impulsive choice in attention-deficit/hyperactivity disorder boys and siblings
Background: impulsive drive for immediate reward (IDIR) and delay aversion are dissociable elements of the preference for immediate over delayed rewards seen in attention-deficit/hyperactivity disorder (ADHD). We hypothesized that IDIR would be associated with dopamine regulating genes and delay aversion would be associated with serotonin-regulating genes.

Methods: impulsive drive for immediate reward and delay aversion were measured in 459 male children and adolescents (328 ADHD and 131 unaffected siblings) with a laboratory choice task. The sample was genotyped for the 5HTT (SLC6A4) promoter serotonin-transporter-linked polymorphic region polymorphism and a DAT1 (SLC6A3) 40-base pair variable number tandem repeat located in the 3?-untranslated region of the gene.

Results: there was no effect of dopamine transporter (DAT)1 on IDIR. As predicted, serotonin-transporter-linked polymorphic region s-allele carriers were more delay averse. This effect was driven by the s/l genotype in the ADHD group. These results were not altered by taking account of the rs25531 A/G single nucleotide polymorphism and were independent of age, IQ, and oppositional defiant disorder symptoms.

Conclusions: the results support the genetic distinctiveness of IDIR and delay aversion in ADHD and implicate serotonin function in delay aversion. Possible explanations of the heterosis effect in the ADHD cases are presented
0006-3223
230-236
Sonuga-Barke, Edmund J.S.
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Kumsta, Robert
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Schlotz, Wolff
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Lasky-Su, Jessica
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Marco, Rafaela
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Miranda, Ana
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Mulas, Fernando
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Oades, Robert D.
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Banaschewski, Tobias
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Mueller, Ueli
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Andreou, Penny
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Christiansen, Hanna
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Gabriels, Isabel
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Uebel, Henrik
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Kuntsi, Jonna
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Franke, Barbara
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Buitelaar, Jan
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Ebstein, Richard
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Gill, Michael
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Anney, Richard
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Roeyers, Herbert
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Rothenberger, Aribert
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Sergeant, Joseph
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Steinhausen, Hans Christoph
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Asherson, Philip
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Faraone, Stephen V.
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Sonuga-Barke, Edmund J.S.
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Kumsta, Robert
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Schlotz, Wolff
49499d5e-4ff4-4ad3-b5f7-eec11b25b5db
Lasky-Su, Jessica
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Marco, Rafaela
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Miranda, Ana
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Mulas, Fernando
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Oades, Robert D.
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Banaschewski, Tobias
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Mueller, Ueli
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Andreou, Penny
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Christiansen, Hanna
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Gabriels, Isabel
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Uebel, Henrik
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Kuntsi, Jonna
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Franke, Barbara
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Buitelaar, Jan
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Ebstein, Richard
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Gill, Michael
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Anney, Richard
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Roeyers, Herbert
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Rothenberger, Aribert
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Sergeant, Joseph
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Steinhausen, Hans Christoph
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Asherson, Philip
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Faraone, Stephen V.
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Sonuga-Barke, Edmund J.S., Kumsta, Robert, Schlotz, Wolff, Lasky-Su, Jessica, Marco, Rafaela, Miranda, Ana, Mulas, Fernando, Oades, Robert D., Banaschewski, Tobias, Mueller, Ueli, Andreou, Penny, Christiansen, Hanna, Gabriels, Isabel, Uebel, Henrik, Kuntsi, Jonna, Franke, Barbara, Buitelaar, Jan, Ebstein, Richard, Gill, Michael, Anney, Richard, Roeyers, Herbert, Rothenberger, Aribert, Sergeant, Joseph, Steinhausen, Hans Christoph, Asherson, Philip and Faraone, Stephen V. (2011) A functional variant of the serotonin transporter gene (SLC6A4) moderates impulsive choice in attention-deficit/hyperactivity disorder boys and siblings. Biological Psychiatry, 70 (3), Summer Issue, 230-236. (doi:10.1016/j.biopsych.2011.01.040). (PMID:21497794)

Record type: Article

Abstract

Background: impulsive drive for immediate reward (IDIR) and delay aversion are dissociable elements of the preference for immediate over delayed rewards seen in attention-deficit/hyperactivity disorder (ADHD). We hypothesized that IDIR would be associated with dopamine regulating genes and delay aversion would be associated with serotonin-regulating genes.

Methods: impulsive drive for immediate reward and delay aversion were measured in 459 male children and adolescents (328 ADHD and 131 unaffected siblings) with a laboratory choice task. The sample was genotyped for the 5HTT (SLC6A4) promoter serotonin-transporter-linked polymorphic region polymorphism and a DAT1 (SLC6A3) 40-base pair variable number tandem repeat located in the 3?-untranslated region of the gene.

Results: there was no effect of dopamine transporter (DAT)1 on IDIR. As predicted, serotonin-transporter-linked polymorphic region s-allele carriers were more delay averse. This effect was driven by the s/l genotype in the ADHD group. These results were not altered by taking account of the rs25531 A/G single nucleotide polymorphism and were independent of age, IQ, and oppositional defiant disorder symptoms.

Conclusions: the results support the genetic distinctiveness of IDIR and delay aversion in ADHD and implicate serotonin function in delay aversion. Possible explanations of the heterosis effect in the ADHD cases are presented

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Published date: August 2011

Identifiers

Local EPrints ID: 193327
URI: http://eprints.soton.ac.uk/id/eprint/193327
ISSN: 0006-3223
PURE UUID: 926b527f-0201-4fda-9450-3e4b750784e8

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Date deposited: 13 Jul 2011 10:53
Last modified: 14 Mar 2024 03:55

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Contributors

Author: Edmund J.S. Sonuga-Barke
Author: Robert Kumsta
Author: Wolff Schlotz
Author: Jessica Lasky-Su
Author: Rafaela Marco
Author: Ana Miranda
Author: Fernando Mulas
Author: Robert D. Oades
Author: Tobias Banaschewski
Author: Ueli Mueller
Author: Penny Andreou
Author: Hanna Christiansen
Author: Isabel Gabriels
Author: Henrik Uebel
Author: Jonna Kuntsi
Author: Barbara Franke
Author: Jan Buitelaar
Author: Richard Ebstein
Author: Michael Gill
Author: Richard Anney
Author: Herbert Roeyers
Author: Aribert Rothenberger
Author: Joseph Sergeant
Author: Hans Christoph Steinhausen
Author: Philip Asherson
Author: Stephen V. Faraone

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