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Identification of four new translocations involving FGFR1 in myeloid disorders

Identification of four new translocations involving FGFR1 in myeloid disorders
Identification of four new translocations involving FGFR1 in myeloid disorders
The 8p11 myeloproliferative syndrome (EMS) is associated with three translocations, t(8;13)(p11;q12), t(8;9)(p11;q33), and t(6;8)(q27;p11), that fuse unrelated genes (ZNF198, CEP110, and FOP, respectively) to the entire tyrosine kinase domain of FGFR1. In all cases thus far examined (n = 10), the t(8;13) results in an identical mRNA fusion between ZNF198 exon 17 and FGFR1 exon 9. To determine if consistent fusions are also seen in the variant translocations, we performed RT-PCR on four cases and sequenced the products. For two patients with a t(8;9), we found that CEP110 exon 15 was fused to FGFR1 exon 9. For two patients with a t(6;8), we found that FOP exon 5 (n = 1) or exon 7 (n = 1) was fused to FGFR1 exon 9. To determine if FGFR1 might be involved in other myeloid disorders with translocations of 8p, we developed a two-color FISH assay using two differentially labeled PAC clones that flank FGFR1. Disruption of this gene was indicated in a patient with a t(8;17)(p11;q25) and Ph-negative chronic myeloid leukemia in association with systemic malignant mast cell disease, a patient with acute myeloid leukemia with a t(8;11)(p11;p15), and two cases with T-cell lymphoma, myeloproliferative disorder, and marrow eosinophilia with a t(8;12)(p11;q15) and ins(12;8)(p11;p11p21), respectively. For the patient with the t(8;11), the chromosome 11 breakpoint was determined to be in the vicinity of NUP98. We conclude that 1) all mRNA fusions in EMS result in splicing to FGFR1 exon 9 but breakpoints in FOP are variable, 2) two-color FISH can identify patients with EMS, and 3) the t(8;17)(p11;q25), t(8;11)(p11;p15), t(8;12)(p11;q15), and ins(12;8)(p11;p11p21) are novel karyotypic changes that most likely involve FGFR1.
1098-2264
155-163
Sohal, Jastinder
6a033b88-2ad9-4899-a745-e15f9ccdfd18
Chase, Andrew
a40a09c2-3073-4655-ba0b-a802e34914b5
Mould, Sarah
1beb42f6-2dc0-4e7e-8922-14d60b08e2d7
Corcoran, Martin
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Oscier, David
2e7f0cc1-93e2-441e-857d-7314efae08ec
Iqbal, Sameena
5466a67e-c498-4a61-8f16-7aef3c81f1c9
Parker, Sally
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Welborn, Jeanna
e198e6dd-03cf-4b25-9c38-56d518f5a08f
Harris, Richard I.
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Martinelli, Giovanni
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Montefusco, Vittorio
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Sinclair, Paul
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Wilkins, Bridget S.
eb7a2bdd-ea12-49a0-9a80-cfe841f1189e
Van den Berg, Henk
26f9ceed-69e7-4790-8236-c5e92096162d
Vanstraelen, Danny
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Goldman, John M.
2632cc83-5866-46bd-914d-8450157fe689
Cross, Nicholas C.P.
f87650da-b908-4a34-b31b-d62c5f186fe4
Sohal, Jastinder
6a033b88-2ad9-4899-a745-e15f9ccdfd18
Chase, Andrew
a40a09c2-3073-4655-ba0b-a802e34914b5
Mould, Sarah
1beb42f6-2dc0-4e7e-8922-14d60b08e2d7
Corcoran, Martin
6538f86c-f243-4b6e-8a4d-2f11db51faf2
Oscier, David
2e7f0cc1-93e2-441e-857d-7314efae08ec
Iqbal, Sameena
5466a67e-c498-4a61-8f16-7aef3c81f1c9
Parker, Sally
6d99d938-18b6-4628-8c6b-91fcd0e3c1e6
Welborn, Jeanna
e198e6dd-03cf-4b25-9c38-56d518f5a08f
Harris, Richard I.
e45a69a0-2549-4a1d-ae3b-902c3f3d9c80
Martinelli, Giovanni
4a5508fd-7178-45ca-90dd-c7692af77776
Montefusco, Vittorio
1463643b-3fc9-412e-ac66-2fd917e407d9
Sinclair, Paul
16c6fcf6-3999-4bdd-9b4c-62106af82c0b
Wilkins, Bridget S.
eb7a2bdd-ea12-49a0-9a80-cfe841f1189e
Van den Berg, Henk
26f9ceed-69e7-4790-8236-c5e92096162d
Vanstraelen, Danny
10c808e2-40ad-4b85-a4b5-a33262923abf
Goldman, John M.
2632cc83-5866-46bd-914d-8450157fe689
Cross, Nicholas C.P.
f87650da-b908-4a34-b31b-d62c5f186fe4

Sohal, Jastinder, Chase, Andrew, Mould, Sarah, Corcoran, Martin, Oscier, David, Iqbal, Sameena, Parker, Sally, Welborn, Jeanna, Harris, Richard I., Martinelli, Giovanni, Montefusco, Vittorio, Sinclair, Paul, Wilkins, Bridget S., Van den Berg, Henk, Vanstraelen, Danny, Goldman, John M. and Cross, Nicholas C.P. (2001) Identification of four new translocations involving FGFR1 in myeloid disorders. Genes, Chromosomes and Cancer, 32 (2), 155-163. (doi:10.1002/gcc.1177).

Record type: Article

Abstract

The 8p11 myeloproliferative syndrome (EMS) is associated with three translocations, t(8;13)(p11;q12), t(8;9)(p11;q33), and t(6;8)(q27;p11), that fuse unrelated genes (ZNF198, CEP110, and FOP, respectively) to the entire tyrosine kinase domain of FGFR1. In all cases thus far examined (n = 10), the t(8;13) results in an identical mRNA fusion between ZNF198 exon 17 and FGFR1 exon 9. To determine if consistent fusions are also seen in the variant translocations, we performed RT-PCR on four cases and sequenced the products. For two patients with a t(8;9), we found that CEP110 exon 15 was fused to FGFR1 exon 9. For two patients with a t(6;8), we found that FOP exon 5 (n = 1) or exon 7 (n = 1) was fused to FGFR1 exon 9. To determine if FGFR1 might be involved in other myeloid disorders with translocations of 8p, we developed a two-color FISH assay using two differentially labeled PAC clones that flank FGFR1. Disruption of this gene was indicated in a patient with a t(8;17)(p11;q25) and Ph-negative chronic myeloid leukemia in association with systemic malignant mast cell disease, a patient with acute myeloid leukemia with a t(8;11)(p11;p15), and two cases with T-cell lymphoma, myeloproliferative disorder, and marrow eosinophilia with a t(8;12)(p11;q15) and ins(12;8)(p11;p11p21), respectively. For the patient with the t(8;11), the chromosome 11 breakpoint was determined to be in the vicinity of NUP98. We conclude that 1) all mRNA fusions in EMS result in splicing to FGFR1 exon 9 but breakpoints in FOP are variable, 2) two-color FISH can identify patients with EMS, and 3) the t(8;17)(p11;q25), t(8;11)(p11;p15), t(8;12)(p11;q15), and ins(12;8)(p11;p11p21) are novel karyotypic changes that most likely involve FGFR1.

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Published date: 2001

Identifiers

Local EPrints ID: 24960
URI: http://eprints.soton.ac.uk/id/eprint/24960
ISSN: 1098-2264
PURE UUID: 9b3f3070-fed3-492c-b56f-4ec91e7f0ec0
ORCID for Andrew Chase: ORCID iD orcid.org/0000-0001-6617-9953
ORCID for Nicholas C.P. Cross: ORCID iD orcid.org/0000-0001-5481-2555

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Date deposited: 05 Apr 2006
Last modified: 16 Mar 2024 03:23

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Contributors

Author: Jastinder Sohal
Author: Andrew Chase ORCID iD
Author: Sarah Mould
Author: Martin Corcoran
Author: David Oscier
Author: Sameena Iqbal
Author: Sally Parker
Author: Jeanna Welborn
Author: Richard I. Harris
Author: Giovanni Martinelli
Author: Vittorio Montefusco
Author: Paul Sinclair
Author: Bridget S. Wilkins
Author: Henk Van den Berg
Author: Danny Vanstraelen
Author: John M. Goldman

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