The University of Southampton
University of Southampton Institutional Repository

Serum insulin-like growth factor (IGF)-I and IGF-binding protein-1 in elderly people: relationships with cardiovascular risk factors, body composition, size at birth, and childhood growth

Serum insulin-like growth factor (IGF)-I and IGF-binding protein-1 in elderly people: relationships with cardiovascular risk factors, body composition, size at birth, and childhood growth
Serum insulin-like growth factor (IGF)-I and IGF-binding protein-1 in elderly people: relationships with cardiovascular risk factors, body composition, size at birth, and childhood growth
The IGF system is important in regulation of fetal and childhood growth. In later life, IGF-I and IGF-binding protein-1 (IGFBP-1) have been implicated in the pathogenesis of arteriosclerosis. They are, thus, potential candidates in explaining the link between early growth and adult cardiovascular disease. We measured fasting serum IGF-I and IGFBP-1 concentrations in 394 men and women from a cohort of 7086 individuals, born between 1924 and 1933 in Helsinki, Finland, whose weight and height were recorded at birth and from 7 to 15 yr of age. They also underwent clinical examination, including measurement of body fat using bioimpedance, blood pressure, glucose tolerance, and plasma insulin and fibrinogen concentrations. Serum IGF-I was positively correlated with fasting glucose (r = 0.10, P = 0.06) and fibrinogen (r = 0.19, P = 0.0001) concentrations and blood pressure (systolic and diastolic r = 0.10, P 0.05) and inversely with percentage body fat (r = -0.13, P = 0.01) and waist circumference (r = -0.11, P = 0.03). IGFBP-1 was inversely correlated with adult body mass index (BMI) (r = -0.46, P < 0.0001), fasting glucose and insulin concentrations, and blood pressure.
There were correlations between the adult level of IGFBP-1 and birth weight (r = 0.11, P = 0.03) and ponderal index (weight/length3) at birth (r = 0.13, P = 0.01), but IGF-I was not related to birth measurements. There were interactive effects between childhood height or BMI and adult BMI on IGF-I and IGFBP-1 in adulthood. Tall height and high BMI at 7 yr were associated with low IGF-I (P = 0.03 for height and P = 0.003 for BMI) and high IGFBP-1 (P = 0.02 and P = 0.06) in adulthood but only in those subjects whose current BMI was below median. On further analysis these interactive effects were particularly strong for height in childhood and adult lean BMI (lean body mass/height2). Among men and women of below-average lean BMI, tall height at 7 yr was associated with low adult IGF-I (P = 0.007) and high IGFBP-1 (P = 0.0004) concentrations [interaction (7-yr height x adult lean BMI); P = 0.008 for IGF-I and 0.001 for IGFBP-1].
There is no evidence that reduced fetal growth programs IGF-I concentrations in old age. An association between small size at birth and low IGFBP-1 concentrations may in part reflect fetal programming effects on insulin resistance. Given the anabolic effects of the GH-IGF-I axis, subjects with tall height in childhood but low adult lean body mass may be at risk of late-life GH-IGF-I axis dysfunction. Prospective studies should address whether this group is susceptible to type 2 diabetes, coronary heart disease, and osteoporosis.
This work was supported by grants from Academy of Finland, British Heart Foundation, Cancer Society of Finland, Finska Läkaresällskapet, Helsinki University Central Hospital Research Fund, The Foundation for Pediatric Research, Sigrid Jusélius Foundation, Sydäntutkimussäätiö, and National Institute of Child Development Grant 1-R01-HD41107-01 (to D.I.W.P.).
0021-972X
1059-1065
Kajantie, Eero
d68d55b6-6df1-4195-a914-44c738a6db93
Fall, Caroline H.
7171a105-34f5-4131-89d7-1aa639893b18
Seppala, Markku
9f5a8324-72a0-41f7-8230-fccf970dc4c9
Koistinen, Riita
aeca03ed-c806-4678-8abb-84069c71b4af
Dunkel, Leo
45b1f986-5178-44bc-8e9b-a815688dadfe
Yliharsila, Hilkka
43f42eb3-57cf-4a49-af0f-6672525f23d5
Osmond, Clive
2677bf85-494f-4a78-adf8-580e1b8acb81
Andersson, Sture
31b08a06-75c5-4aa5-b879-3cb270abe81f
Barker, David J.P.
5c773838-b094-4ac1-999b-b5869717f243
Forsen, Tom
77245dbe-fc1f-4e6f-a2bb-6f1a77918cd2
Holt, Richard I.G.
d54202e1-fcf6-4a17-a320-9f32d7024393
Phillips, David I.W.
29b73be7-2ff9-4fff-ae42-d59842df4cc6
Eriksson, Johan
bed81786-e72a-4710-a5b7-ddc6732bc45a
Kajantie, Eero
d68d55b6-6df1-4195-a914-44c738a6db93
Fall, Caroline H.
7171a105-34f5-4131-89d7-1aa639893b18
Seppala, Markku
9f5a8324-72a0-41f7-8230-fccf970dc4c9
Koistinen, Riita
aeca03ed-c806-4678-8abb-84069c71b4af
Dunkel, Leo
45b1f986-5178-44bc-8e9b-a815688dadfe
Yliharsila, Hilkka
43f42eb3-57cf-4a49-af0f-6672525f23d5
Osmond, Clive
2677bf85-494f-4a78-adf8-580e1b8acb81
Andersson, Sture
31b08a06-75c5-4aa5-b879-3cb270abe81f
Barker, David J.P.
5c773838-b094-4ac1-999b-b5869717f243
Forsen, Tom
77245dbe-fc1f-4e6f-a2bb-6f1a77918cd2
Holt, Richard I.G.
d54202e1-fcf6-4a17-a320-9f32d7024393
Phillips, David I.W.
29b73be7-2ff9-4fff-ae42-d59842df4cc6
Eriksson, Johan
bed81786-e72a-4710-a5b7-ddc6732bc45a

Kajantie, Eero, Fall, Caroline H., Seppala, Markku, Koistinen, Riita, Dunkel, Leo, Yliharsila, Hilkka, Osmond, Clive, Andersson, Sture, Barker, David J.P., Forsen, Tom, Holt, Richard I.G., Phillips, David I.W. and Eriksson, Johan (2003) Serum insulin-like growth factor (IGF)-I and IGF-binding protein-1 in elderly people: relationships with cardiovascular risk factors, body composition, size at birth, and childhood growth. Journal of Clinical Endocrinology & Metabolism, 88 (3), 1059-1065. (doi:10.1210/jc.2002-021380).

Record type: Article

Abstract

The IGF system is important in regulation of fetal and childhood growth. In later life, IGF-I and IGF-binding protein-1 (IGFBP-1) have been implicated in the pathogenesis of arteriosclerosis. They are, thus, potential candidates in explaining the link between early growth and adult cardiovascular disease. We measured fasting serum IGF-I and IGFBP-1 concentrations in 394 men and women from a cohort of 7086 individuals, born between 1924 and 1933 in Helsinki, Finland, whose weight and height were recorded at birth and from 7 to 15 yr of age. They also underwent clinical examination, including measurement of body fat using bioimpedance, blood pressure, glucose tolerance, and plasma insulin and fibrinogen concentrations. Serum IGF-I was positively correlated with fasting glucose (r = 0.10, P = 0.06) and fibrinogen (r = 0.19, P = 0.0001) concentrations and blood pressure (systolic and diastolic r = 0.10, P 0.05) and inversely with percentage body fat (r = -0.13, P = 0.01) and waist circumference (r = -0.11, P = 0.03). IGFBP-1 was inversely correlated with adult body mass index (BMI) (r = -0.46, P < 0.0001), fasting glucose and insulin concentrations, and blood pressure.
There were correlations between the adult level of IGFBP-1 and birth weight (r = 0.11, P = 0.03) and ponderal index (weight/length3) at birth (r = 0.13, P = 0.01), but IGF-I was not related to birth measurements. There were interactive effects between childhood height or BMI and adult BMI on IGF-I and IGFBP-1 in adulthood. Tall height and high BMI at 7 yr were associated with low IGF-I (P = 0.03 for height and P = 0.003 for BMI) and high IGFBP-1 (P = 0.02 and P = 0.06) in adulthood but only in those subjects whose current BMI was below median. On further analysis these interactive effects were particularly strong for height in childhood and adult lean BMI (lean body mass/height2). Among men and women of below-average lean BMI, tall height at 7 yr was associated with low adult IGF-I (P = 0.007) and high IGFBP-1 (P = 0.0004) concentrations [interaction (7-yr height x adult lean BMI); P = 0.008 for IGF-I and 0.001 for IGFBP-1].
There is no evidence that reduced fetal growth programs IGF-I concentrations in old age. An association between small size at birth and low IGFBP-1 concentrations may in part reflect fetal programming effects on insulin resistance. Given the anabolic effects of the GH-IGF-I axis, subjects with tall height in childhood but low adult lean body mass may be at risk of late-life GH-IGF-I axis dysfunction. Prospective studies should address whether this group is susceptible to type 2 diabetes, coronary heart disease, and osteoporosis.
This work was supported by grants from Academy of Finland, British Heart Foundation, Cancer Society of Finland, Finska Läkaresällskapet, Helsinki University Central Hospital Research Fund, The Foundation for Pediatric Research, Sigrid Jusélius Foundation, Sydäntutkimussäätiö, and National Institute of Child Development Grant 1-R01-HD41107-01 (to D.I.W.P.).

This record has no associated files available for download.

More information

Published date: 2003

Identifiers

Local EPrints ID: 25686
URI: http://eprints.soton.ac.uk/id/eprint/25686
ISSN: 0021-972X
PURE UUID: 0c6553d8-7c78-4e95-b82f-272a172099fb
ORCID for Caroline H. Fall: ORCID iD orcid.org/0000-0003-4402-5552
ORCID for Clive Osmond: ORCID iD orcid.org/0000-0002-9054-4655
ORCID for Richard I.G. Holt: ORCID iD orcid.org/0000-0001-8911-6744

Catalogue record

Date deposited: 21 Apr 2006
Last modified: 16 Mar 2024 03:19

Export record

Altmetrics

Contributors

Author: Eero Kajantie
Author: Markku Seppala
Author: Riita Koistinen
Author: Leo Dunkel
Author: Hilkka Yliharsila
Author: Clive Osmond ORCID iD
Author: Sture Andersson
Author: David J.P. Barker
Author: Tom Forsen
Author: David I.W. Phillips
Author: Johan Eriksson

Download statistics

Downloads from ePrints over the past year. Other digital versions may also be available to download e.g. from the publisher's website.

View more statistics

Atom RSS 1.0 RSS 2.0

Contact ePrints Soton: eprints@soton.ac.uk

ePrints Soton supports OAI 2.0 with a base URL of http://eprints.soton.ac.uk/cgi/oai2

This repository has been built using EPrints software, developed at the University of Southampton, but available to everyone to use.

We use cookies to ensure that we give you the best experience on our website. If you continue without changing your settings, we will assume that you are happy to receive cookies on the University of Southampton website.

×