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Activation markers of human basophils: CD69 expression is strongly and preferentially induced by IL-3

Activation markers of human basophils: CD69 expression is strongly and preferentially induced by IL-3
Activation markers of human basophils: CD69 expression is strongly and preferentially induced by IL-3
Background: The biological functions of basophils are precisely regulated by various cytokines in vitro, but little is known about surface markers that are upregulated during the cytokine-mediated activation process.
Objective: It has been well established that CD69, CD44, and CD54 represent "activation markers" for cytokine-mediated eosinophil activation. The objective of this study was to elucidate the expression and regulation of these molecules in human basophils in vitro as well as in vivo.
Methods: Basophils were purified from venous blood by means of density gradient centrifugation followed by negative selection. Surface expression was analyzed by means of flow cytometry. We also studied the expression of CD69, CD44, and CD54 on basophils in bronchoalveolar lavage fluid and blood specimens from patients with asthma.
Results: CD44 and CD54 were constitutively expressed on basophils and moderately upregulated by IL-3. On the other hand, CD69 expression was only weakly observed in freshly isolated basophils, but IL-3 induced extremely high levels of expression. Surface CD69 appeared rather slowly in comparison with CD63 and CD11b, and the induction of expression was completed within 24 hours. Basophil CD69 had no functional relevance, but it did have biological relevance. Whole blood basophils from asthmatic individuals expressed significantly higher levels of CD69 than did those from normal individuals. Furthermore, bronchoalveolar lavage fluid basophils showed higher levels of CD69 expression than did blood basophils from the same donors.
Conclusion: CD69 expression on basophils was preferentially and strongly upregulated by IL-3. CD69 on basophils might be useful as an in vitro as well as in vivo marker of activation of these cells by IL-3.
Basophils, eosinophils, allergy, bronchial asthma, cytokines, bronchoalveolar lavage fluid, CD69, IL-3, human
0091-6749
817-823
Yoshimura, Chitose
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Yamaguchi, Masao
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Iikura, Motoyasu
1c3c324c-18b3-45fa-95c9-03f4fa68f02a
Izumi, Shinyu
a6b9e689-1778-40a8-89c7-035e409154b5
Kudo, Koichiro
d3427ab2-0af3-4efa-bf75-b010c59cbe72
Nagase, Hiroyuki
ea79aa89-a811-430b-8be1-37d5e85149cc
Ishii, Akira
2ab9f22f-ece5-4aaa-878b-d3143447b416
Walls, Andrew F.
aaa7e455-0562-4b4c-94f5-ec29c74b1bfe
Ra, Chisei
fa9ef555-9780-481f-9b44-589c074863d2
Iwata, Tsutomu
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Igarashi, Takashi
d1c48765-3f2c-4741-9292-a30fe2c63723
Yamamoto, Kazuhiko
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Hirai, Koichi
c0a43de9-aed8-4f34-b011-7232daf10c54
Yoshimura, Chitose
a8458a63-2d48-4b48-8496-e4e3f7e5c665
Yamaguchi, Masao
b3db1282-6d9c-4b4c-9647-4de0fe6dc365
Iikura, Motoyasu
1c3c324c-18b3-45fa-95c9-03f4fa68f02a
Izumi, Shinyu
a6b9e689-1778-40a8-89c7-035e409154b5
Kudo, Koichiro
d3427ab2-0af3-4efa-bf75-b010c59cbe72
Nagase, Hiroyuki
ea79aa89-a811-430b-8be1-37d5e85149cc
Ishii, Akira
2ab9f22f-ece5-4aaa-878b-d3143447b416
Walls, Andrew F.
aaa7e455-0562-4b4c-94f5-ec29c74b1bfe
Ra, Chisei
fa9ef555-9780-481f-9b44-589c074863d2
Iwata, Tsutomu
8e37398a-8edd-4d9f-8d9a-0aff97bf37dd
Igarashi, Takashi
d1c48765-3f2c-4741-9292-a30fe2c63723
Yamamoto, Kazuhiko
98495915-f02d-47d5-9f09-6b588455df7d
Hirai, Koichi
c0a43de9-aed8-4f34-b011-7232daf10c54

Yoshimura, Chitose, Yamaguchi, Masao, Iikura, Motoyasu, Izumi, Shinyu, Kudo, Koichiro, Nagase, Hiroyuki, Ishii, Akira, Walls, Andrew F., Ra, Chisei, Iwata, Tsutomu, Igarashi, Takashi, Yamamoto, Kazuhiko and Hirai, Koichi (2002) Activation markers of human basophils: CD69 expression is strongly and preferentially induced by IL-3. Journal of Allergy and Clinical Immunology, 109 (5), 817-823. (doi:10.1067/mai.2002.123532).

Record type: Article

Abstract

Background: The biological functions of basophils are precisely regulated by various cytokines in vitro, but little is known about surface markers that are upregulated during the cytokine-mediated activation process.
Objective: It has been well established that CD69, CD44, and CD54 represent "activation markers" for cytokine-mediated eosinophil activation. The objective of this study was to elucidate the expression and regulation of these molecules in human basophils in vitro as well as in vivo.
Methods: Basophils were purified from venous blood by means of density gradient centrifugation followed by negative selection. Surface expression was analyzed by means of flow cytometry. We also studied the expression of CD69, CD44, and CD54 on basophils in bronchoalveolar lavage fluid and blood specimens from patients with asthma.
Results: CD44 and CD54 were constitutively expressed on basophils and moderately upregulated by IL-3. On the other hand, CD69 expression was only weakly observed in freshly isolated basophils, but IL-3 induced extremely high levels of expression. Surface CD69 appeared rather slowly in comparison with CD63 and CD11b, and the induction of expression was completed within 24 hours. Basophil CD69 had no functional relevance, but it did have biological relevance. Whole blood basophils from asthmatic individuals expressed significantly higher levels of CD69 than did those from normal individuals. Furthermore, bronchoalveolar lavage fluid basophils showed higher levels of CD69 expression than did blood basophils from the same donors.
Conclusion: CD69 expression on basophils was preferentially and strongly upregulated by IL-3. CD69 on basophils might be useful as an in vitro as well as in vivo marker of activation of these cells by IL-3.

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More information

Published date: 2002
Additional Information: Mechanisms of allergy
Keywords: Basophils, eosinophils, allergy, bronchial asthma, cytokines, bronchoalveolar lavage fluid, CD69, IL-3, human

Identifiers

Local EPrints ID: 28642
URI: http://eprints.soton.ac.uk/id/eprint/28642
ISSN: 0091-6749
PURE UUID: 8c5fdd72-62cf-4672-93bf-c36076ea79fd
ORCID for Andrew F. Walls: ORCID iD orcid.org/0000-0003-4803-4595

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Date deposited: 02 May 2006
Last modified: 16 Mar 2024 02:38

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Contributors

Author: Chitose Yoshimura
Author: Masao Yamaguchi
Author: Motoyasu Iikura
Author: Shinyu Izumi
Author: Koichiro Kudo
Author: Hiroyuki Nagase
Author: Akira Ishii
Author: Andrew F. Walls ORCID iD
Author: Chisei Ra
Author: Tsutomu Iwata
Author: Takashi Igarashi
Author: Kazuhiko Yamamoto
Author: Koichi Hirai

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