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Genome-wide association study to identify genetic determinants of severe asthma

Genome-wide association study to identify genetic determinants of severe asthma
Genome-wide association study to identify genetic determinants of severe asthma
Background The genetic basis for developing asthma has been extensively studied. However, association studies to date have mostly focused on mild to moderate disease and genetic risk factors for severe asthma remain unclear.

Objective To identify common genetic variants affecting susceptibility to severe asthma.

Methods A genome-wide association study was undertaken in 933 European ancestry individuals with severe asthma based on Global Initiative for Asthma (GINA) criteria 3 or above and 3346 clean controls. After standard quality control measures, the association of 480?889 genotyped single nucleotide polymorphisms (SNPs) was tested. To improve the resolution of the association signals identified, non-genotyped SNPs were imputed in these regions using a dense reference panel of SNP genotypes from the 1000 Genomes Project. Then replication of SNPs of interest was undertaken in a further 231 cases and 1345 controls and a meta-analysis was performed to combine the results across studies.

Results An association was confirmed in subjects with severe asthma of loci previously identified for association with mild to moderate asthma. The strongest evidence was seen for the ORMDL3/GSDMB locus on chromosome 17q12-21 (rs4794820, p=1.03×10(?8) following meta-analysis) meeting genome-wide significance. Strong evidence was also found for the IL1RL1/IL18R1 locus on 2q12 (rs9807989, p=5.59×10(?8) following meta-analysis) just below this threshold. No novel loci for susceptibility to severe asthma met strict criteria for genome-wide significance.

Conclusions The largest genome-wide association study of severe asthma to date was carried out and strong evidence found for the association of two previously identified asthma susceptibility loci in patients with severe disease. A number of novel regions with suggestive evidence were also identified warranting further study.

0040-6376
762-768
Wan, Y. I.
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Shrine, N. R. G.
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Soler Artigas, M.
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Wain, L. V.
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Blakey, J. D.
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Moffatt, M. F.
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Bush, A.
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Chung, K. F.
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Cookson, W. O. C. M.
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Strachan, D. P.
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Heaney, L.
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Al-Momani, B. A. H.
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Mansur, A. H.
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Manney, S.
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Thomson, N. C.
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Chaudhuri, R.
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Brightling, C. E.
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Bafadhel, M.
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Niven, R.
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Simpson, A.
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Holloway, J. W.
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Howarth, P. H.
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Hui, J.
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Musk, A. W.
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Baltic, S.
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Ferreira, M. A. R.
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Thompson, P. J.
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Tobin, M. D.
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Sayers, I.
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Hall, I. P.
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Wan, Y. I.
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Shrine, N. R. G.
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Soler Artigas, M.
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Strachan, D. P.
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Heaney, L.
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Al-Momani, B. A. H.
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Mansur, A. H.
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Manney, S.
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Thomson, N. C.
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Chaudhuri, R.
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Bafadhel, M.
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Singapuri, A.
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Niven, R.
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Holloway, J. W.
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Howarth, P. H.
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Hui, J.
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James, A. L.
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Tobin, M. D.
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Sayers, I.
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Hall, I. P.
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Wan, Y. I., Shrine, N. R. G., Soler Artigas, M., Wain, L. V., Blakey, J. D., Moffatt, M. F., Bush, A., Chung, K. F., Cookson, W. O. C. M., Strachan, D. P., Heaney, L., Al-Momani, B. A. H., Mansur, A. H., Manney, S., Thomson, N. C., Chaudhuri, R., Brightling, C. E., Bafadhel, M., Singapuri, A., Niven, R., Simpson, A., Holloway, J. W., Howarth, P. H., Hui, J., Musk, A. W., James, A. L., Brown, M. A., Baltic, S., Ferreira, M. A. R., Thompson, P. J., Tobin, M. D., Sayers, I. and Hall, I. P. (2012) Genome-wide association study to identify genetic determinants of severe asthma. Thorax, 67 (9), 762-768. (doi:10.1136/thoraxjnl-2011-201262).

Record type: Article

Abstract

Background The genetic basis for developing asthma has been extensively studied. However, association studies to date have mostly focused on mild to moderate disease and genetic risk factors for severe asthma remain unclear.

Objective To identify common genetic variants affecting susceptibility to severe asthma.

Methods A genome-wide association study was undertaken in 933 European ancestry individuals with severe asthma based on Global Initiative for Asthma (GINA) criteria 3 or above and 3346 clean controls. After standard quality control measures, the association of 480?889 genotyped single nucleotide polymorphisms (SNPs) was tested. To improve the resolution of the association signals identified, non-genotyped SNPs were imputed in these regions using a dense reference panel of SNP genotypes from the 1000 Genomes Project. Then replication of SNPs of interest was undertaken in a further 231 cases and 1345 controls and a meta-analysis was performed to combine the results across studies.

Results An association was confirmed in subjects with severe asthma of loci previously identified for association with mild to moderate asthma. The strongest evidence was seen for the ORMDL3/GSDMB locus on chromosome 17q12-21 (rs4794820, p=1.03×10(?8) following meta-analysis) meeting genome-wide significance. Strong evidence was also found for the IL1RL1/IL18R1 locus on 2q12 (rs9807989, p=5.59×10(?8) following meta-analysis) just below this threshold. No novel loci for susceptibility to severe asthma met strict criteria for genome-wide significance.

Conclusions The largest genome-wide association study of severe asthma to date was carried out and strong evidence found for the association of two previously identified asthma susceptibility loci in patients with severe disease. A number of novel regions with suggestive evidence were also identified warranting further study.

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More information

Accepted/In Press date: 5 May 2012
Published date: September 2012
Organisations: Human Development & Health, Clinical & Experimental Sciences

Identifiers

Local EPrints ID: 338176
URI: http://eprints.soton.ac.uk/id/eprint/338176
ISSN: 0040-6376
PURE UUID: bbdf5452-9178-4c9a-b7cf-930288add96a
ORCID for J. W. Holloway: ORCID iD orcid.org/0000-0001-9998-0464

Catalogue record

Date deposited: 10 May 2012 13:45
Last modified: 15 Mar 2024 02:56

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Contributors

Author: Y. I. Wan
Author: N. R. G. Shrine
Author: M. Soler Artigas
Author: L. V. Wain
Author: J. D. Blakey
Author: M. F. Moffatt
Author: A. Bush
Author: K. F. Chung
Author: W. O. C. M. Cookson
Author: D. P. Strachan
Author: L. Heaney
Author: B. A. H. Al-Momani
Author: A. H. Mansur
Author: S. Manney
Author: N. C. Thomson
Author: R. Chaudhuri
Author: C. E. Brightling
Author: M. Bafadhel
Author: A. Singapuri
Author: R. Niven
Author: A. Simpson
Author: J. W. Holloway ORCID iD
Author: P. H. Howarth
Author: J. Hui
Author: A. W. Musk
Author: A. L. James
Author: M. A. Brown
Author: S. Baltic
Author: M. A. R. Ferreira
Author: P. J. Thompson
Author: M. D. Tobin
Author: I. Sayers
Author: I. P. Hall

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