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Antidepressant medications and osteoporosis

Antidepressant medications and osteoporosis
Antidepressant medications and osteoporosis
Use of antidepressant medications that act on the serotonin system has been linked to detrimental impacts on bone mineral density (BMD), and to osteoporosis. This article reviews current evidence for such effects, and identifies themes for future research. Serotonin receptors are found in all major types of bone cell (osteoblasts, osteocytes, and osteoclasts), indicating an important role of the neuroendocrine system in bone. Observational studies indicate a complex relationship between depression, antidepressants, and fracture. First, the presence of depression itself increases fracture risk, in relation with decreased BMD and an increase in falls. A range of aspects of depression may operate, including behavioral factors (e.g., smoking and nutrition), biological changes, and confounders (e.g., comorbidities and concomitant medications). A substantial proportion of depressed patients receive antidepressants, mostly selective serotonin reuptake inhibitors (SSRIs). Some of these have been linked to decreased BMD (SSRIs) and increased fracture risk (SSRIs and tricyclic agents). Current use of SSRIs and tricyclics increases fracture risk by as much as twofold versus nonusers, even after adjustment for potential confounders. While there is a dose–response relationship for SSRIs, the effect does not appear to be homogeneous across the whole class of drugs and may be linked to affinity for the serotonin transporter system. The increase in risk is the greatest in the early stages of treatment, with a dramatic increase after initiation, reaching a peak within 1 month for tricyclics and 8 months for SSRIs. Treatment-associated increased risk diminishes towards baseline in the year following discontinuation. The body of evidence suggests that SSRIs should be considered in the list of medications that are risk factors for osteoporotic fractures
antidepressants, bone mineral density, depression, fracture, osteoporosis, serotonin
8756-3282
606-613
Rizzoli, R.
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Cooper, C.
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Reginster, J.Y.
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Abrahamsen, B.
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Adachi, J.
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Brandi, M.L.
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Bruyere, O.
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Compston, J.
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Ducy, P.
d2c465da-4c7e-494c-8152-3a37fbda1845
Ferrari, S.
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Harvey, N.C.
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Kanis, J.A.
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Karsenty, G.
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Laslop, A.
ca0887e1-5fce-4ac3-bbaf-f2aa98be3e0f
Rabenda, V.
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Vestergaard, P.
b00ba0f8-a9b7-45f1-a0c7-579fec88b556
Rizzoli, R.
2214fb77-8fb7-4c0b-bfc4-9f8d3cace5d7
Cooper, C.
e05f5612-b493-4273-9b71-9e0ce32bdad6
Reginster, J.Y.
4083b457-5347-4ece-a53e-af19c8868c42
Abrahamsen, B.
fee8b1eb-c267-4d2a-952a-d1b9f20d0125
Adachi, J.
89aeb1f8-9966-4630-b5c1-cd8314b20800
Brandi, M.L.
4147f967-9c22-494d-ae35-c8141161413d
Bruyere, O.
9c455ea4-7f77-407f-ab5a-ecb571db8245
Compston, J.
b64c0d0e-97dd-44c8-97ba-f756f0bc966d
Ducy, P.
d2c465da-4c7e-494c-8152-3a37fbda1845
Ferrari, S.
6746194f-54f1-4c75-bcec-a7fcf2bc0c8b
Harvey, N.C.
ce487fb4-d360-4aac-9d17-9466d6cba145
Kanis, J.A.
8da04a36-08a7-4310-b4b4-a6d432439587
Karsenty, G.
6dad0320-c657-4f62-a07f-3a1ff5b60d0b
Laslop, A.
ca0887e1-5fce-4ac3-bbaf-f2aa98be3e0f
Rabenda, V.
031d104e-c09a-4802-9cc6-13112ca20e9c
Vestergaard, P.
b00ba0f8-a9b7-45f1-a0c7-579fec88b556

Rizzoli, R., Cooper, C., Reginster, J.Y., Abrahamsen, B., Adachi, J., Brandi, M.L., Bruyere, O., Compston, J., Ducy, P., Ferrari, S., Harvey, N.C., Kanis, J.A., Karsenty, G., Laslop, A., Rabenda, V. and Vestergaard, P. (2012) Antidepressant medications and osteoporosis. Bone, 51, 606-613. (doi:10.1016/j.bone.2012.05.018). (PMID:22659406)

Record type: Article

Abstract

Use of antidepressant medications that act on the serotonin system has been linked to detrimental impacts on bone mineral density (BMD), and to osteoporosis. This article reviews current evidence for such effects, and identifies themes for future research. Serotonin receptors are found in all major types of bone cell (osteoblasts, osteocytes, and osteoclasts), indicating an important role of the neuroendocrine system in bone. Observational studies indicate a complex relationship between depression, antidepressants, and fracture. First, the presence of depression itself increases fracture risk, in relation with decreased BMD and an increase in falls. A range of aspects of depression may operate, including behavioral factors (e.g., smoking and nutrition), biological changes, and confounders (e.g., comorbidities and concomitant medications). A substantial proportion of depressed patients receive antidepressants, mostly selective serotonin reuptake inhibitors (SSRIs). Some of these have been linked to decreased BMD (SSRIs) and increased fracture risk (SSRIs and tricyclic agents). Current use of SSRIs and tricyclics increases fracture risk by as much as twofold versus nonusers, even after adjustment for potential confounders. While there is a dose–response relationship for SSRIs, the effect does not appear to be homogeneous across the whole class of drugs and may be linked to affinity for the serotonin transporter system. The increase in risk is the greatest in the early stages of treatment, with a dramatic increase after initiation, reaching a peak within 1 month for tricyclics and 8 months for SSRIs. Treatment-associated increased risk diminishes towards baseline in the year following discontinuation. The body of evidence suggests that SSRIs should be considered in the list of medications that are risk factors for osteoporotic fractures

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More information

Published date: September 2012
Keywords: antidepressants, bone mineral density, depression, fracture, osteoporosis, serotonin
Organisations: Faculty of Health Sciences

Identifiers

Local EPrints ID: 346258
URI: http://eprints.soton.ac.uk/id/eprint/346258
ISSN: 8756-3282
PURE UUID: 29ad4e43-23d7-4c25-92e0-6d4faa582148
ORCID for C. Cooper: ORCID iD orcid.org/0000-0003-3510-0709
ORCID for N.C. Harvey: ORCID iD orcid.org/0000-0002-8194-2512

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Date deposited: 17 Dec 2012 16:07
Last modified: 18 Mar 2024 02:58

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Contributors

Author: R. Rizzoli
Author: C. Cooper ORCID iD
Author: J.Y. Reginster
Author: B. Abrahamsen
Author: J. Adachi
Author: M.L. Brandi
Author: O. Bruyere
Author: J. Compston
Author: P. Ducy
Author: S. Ferrari
Author: N.C. Harvey ORCID iD
Author: J.A. Kanis
Author: G. Karsenty
Author: A. Laslop
Author: V. Rabenda
Author: P. Vestergaard

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