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Malaria systems immunology: Plasmodium vivax induces tolerance during primary infection through dysregulation of neutrophils and dendritic cells

Malaria systems immunology: Plasmodium vivax induces tolerance during primary infection through dysregulation of neutrophils and dendritic cells
Malaria systems immunology: Plasmodium vivax induces tolerance during primary infection through dysregulation of neutrophils and dendritic cells

Objectives

To dissect the transcriptional networks underpinning immune cells responses during primary Plasmodium vivax infection of healthy human adults.

Methods

We conducted network co-expression analysis of next-generation RNA sequencing data from whole blood from P. vivax and P. falciparum controlled human malaria infection (CHMI) of healthy naïve and malaria-exposed volunteers. Single cell transcription signatures were used to deconvolute the bulk RNA-Seq data into cell-specific signals.

Results

Initial exposure to P. vivax induced activation of innate immunity, including efficient antigen presentation and complement activation. However, this effect was accompanied by strong immunosuppression mediated by dendritic cells via the induction of Indoleamine 2,3-Dioxygenase 1(IDO1) and Lymphocyte Activation Gene 3 (LAG3). Additionally, P. vivax induced depletion of neutrophil populations associated with down regulation of 3G-protein coupled receptors, CRXCR1, CXCR2 and CSF3R. Accordingly, in malaria-exposed volunteers the inflammatory response was attenuated, with a decreased class II antigen presentation in dendritic cells. While the immunosuppressive signalling was maintained between plasmodium species, response to P. falciparum was significantly more immunogenic.

Conclusions

In silico analyses suggest that primary infection with P. vivax induces potent immunosuppression mediated by dendritic cells, conditioning subsequent anti-malarial immune responses. Targeting immune evasion mechanisms could be an effective alternative for improving vaccine efficacy.

malaria, , controlled human malaria infection, transcriptomics , dendritic cells (DC), immunoregulation, Vaccination
0163-4453
440-447
Vallejo Pulido, Andres
27bc0b94-0c40-4fd1-9533-7e267d588c0a
Read, Robert
b5caca7b-0063-438a-b703-7ecbb6fc2b51
Arevalo-Herrera, Myriam
1a7e864f-13a1-451f-adf4-60c4797fb820
Herrera, Socrates
15ee87a3-93d7-4ff0-9bba-cf45a3b68756
Elliott, Timothy
16670fa8-c2f9-477a-91df-7c9e5b453e0e
Polak, Marta
e0ac5e1a-7074-4776-ba23-490bd4da612d
Vallejo Pulido, Andres
27bc0b94-0c40-4fd1-9533-7e267d588c0a
Read, Robert
b5caca7b-0063-438a-b703-7ecbb6fc2b51
Arevalo-Herrera, Myriam
1a7e864f-13a1-451f-adf4-60c4797fb820
Herrera, Socrates
15ee87a3-93d7-4ff0-9bba-cf45a3b68756
Elliott, Timothy
16670fa8-c2f9-477a-91df-7c9e5b453e0e
Polak, Marta
e0ac5e1a-7074-4776-ba23-490bd4da612d

Vallejo Pulido, Andres, Read, Robert, Arevalo-Herrera, Myriam, Herrera, Socrates, Elliott, Timothy and Polak, Marta (2018) Malaria systems immunology: Plasmodium vivax induces tolerance during primary infection through dysregulation of neutrophils and dendritic cells. Journal of Infection, 77 (5), 440-447. (doi:10.1016/j.jinf.2018.09.005).

Record type: Article

Abstract

Objectives

To dissect the transcriptional networks underpinning immune cells responses during primary Plasmodium vivax infection of healthy human adults.

Methods

We conducted network co-expression analysis of next-generation RNA sequencing data from whole blood from P. vivax and P. falciparum controlled human malaria infection (CHMI) of healthy naïve and malaria-exposed volunteers. Single cell transcription signatures were used to deconvolute the bulk RNA-Seq data into cell-specific signals.

Results

Initial exposure to P. vivax induced activation of innate immunity, including efficient antigen presentation and complement activation. However, this effect was accompanied by strong immunosuppression mediated by dendritic cells via the induction of Indoleamine 2,3-Dioxygenase 1(IDO1) and Lymphocyte Activation Gene 3 (LAG3). Additionally, P. vivax induced depletion of neutrophil populations associated with down regulation of 3G-protein coupled receptors, CRXCR1, CXCR2 and CSF3R. Accordingly, in malaria-exposed volunteers the inflammatory response was attenuated, with a decreased class II antigen presentation in dendritic cells. While the immunosuppressive signalling was maintained between plasmodium species, response to P. falciparum was significantly more immunogenic.

Conclusions

In silico analyses suggest that primary infection with P. vivax induces potent immunosuppression mediated by dendritic cells, conditioning subsequent anti-malarial immune responses. Targeting immune evasion mechanisms could be an effective alternative for improving vaccine efficacy.

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More information

Accepted/In Press date: 15 September 2018
e-pub ahead of print date: 22 September 2018
Published date: November 2018
Keywords: malaria, , controlled human malaria infection, transcriptomics , dendritic cells (DC), immunoregulation, Vaccination

Identifiers

Local EPrints ID: 423692
URI: http://eprints.soton.ac.uk/id/eprint/423692
ISSN: 0163-4453
PURE UUID: c5026f2f-46e6-41c2-b14b-43ec1baf8837
ORCID for Andres Vallejo Pulido: ORCID iD orcid.org/0000-0002-4688-0598
ORCID for Robert Read: ORCID iD orcid.org/0000-0002-4297-6728
ORCID for Timothy Elliott: ORCID iD orcid.org/0000-0003-1097-0222

Catalogue record

Date deposited: 27 Sep 2018 16:30
Last modified: 06 Jun 2024 04:06

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Contributors

Author: Andres Vallejo Pulido ORCID iD
Author: Robert Read ORCID iD
Author: Myriam Arevalo-Herrera
Author: Socrates Herrera
Author: Timothy Elliott ORCID iD
Author: Marta Polak

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