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Evaluating the effects of the novel GLP-1 analogue liraglutide in Alzheimer’s disease: study protocol for a randomised controlled trial (ELAD study)

Evaluating the effects of the novel GLP-1 analogue liraglutide in Alzheimer’s disease: study protocol for a randomised controlled trial (ELAD study)
Evaluating the effects of the novel GLP-1 analogue liraglutide in Alzheimer’s disease: study protocol for a randomised controlled trial (ELAD study)
Background
Liraglutide is a glucagon-like peptide-1 (GLP-1) analogue currently approved for type 2 diabetes and obesity. Preclinical evidence in transgenic models of Alzheimer’s disease suggests that liraglutide exerts neuroprotective effects by reducing amyloid oligomers, normalising synaptic plasticity and cerebral glucose uptake, and increasing the proliferation of neuronal progenitor cells. The primary objective of the study is to evaluate the change in cerebral glucose metabolic rate after 12 months of treatment with liraglutide in participants with Alzheimer’s disease compared to those who are receiving placebo.

Methods/design
ELAD is a 12-month, multi-centre, randomised, double-blind, placebo-controlled, phase IIb trial of liraglutide in participants with mild Alzheimer’s dementia. A total of 206 participants will be randomised to receive either liraglutide or placebo as a daily injection for a year. The primary outcome will be the change in cerebral glucose metabolic rate in the cortical regions (hippocampus, medial temporal lobe, and posterior cingulate) from baseline to follow-up in the treatment group compared with the placebo group. The key secondary outcomes are the change from baseline to 12 months in z scores for clinical and cognitive measures (Alzheimer’s Disease Assessment Scale—Cognitive Subscale and Executive domain scores of the Neuropsychological Test Battery, Clinical Dementia Rating Sum of Boxes, and Alzheimer’s Disease Cooperative Study—Activities of Daily Living) and the incidence and severity of treatment-emergent adverse events or clinically important changes in safety assessments. Other secondary outcomes are 12-month change in magnetic resonance imaging volume, diffusion tensor imaging parameters, reduction in microglial activation in a subgroup of participants, reduction in tau formation and change in amyloid levels in a subgroup of participants measured by tau and amyloid imaging, and changes in composite scores using support machine vector analysis in the treatment group compared with the placebo group.

Discussion
Alzheimer’s disease is a leading cause of morbidity worldwide. As available treatments are only symptomatic, the search for disease-modifying therapies is a priority. If the ELAD trial is successful, liraglutide and GLP-1 analogues will represent an important class of compounds to be further evaluated in clinical trials for Alzheimer’s treatment.

Trial registration
ClinicalTrials.gov, NCT01843075. Registration 30 April 2013.
1745-6215
Femminella, Grazia Daniela
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Frangou, Eleni
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Love, Sharon B.
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Busza, Gail
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Holmes, Clive
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Lawrence, Robert
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Mcfarlane, Brady
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Tadros, George
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Ridha, Basil H.
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Bannister, Carol
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Walker, Zuzana
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Archer, Hilary
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Coulthard, Elizabeth
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Underwood, Ben R.
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Prasanna, Aparna
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Nilforooshan, Ramin
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Thacker, Simon
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Knight, Lucy
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Harrison, John
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Edison, Paul
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Frangou, Eleni
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Busza, Gail
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Holmes, Clive
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Malik, Naghma
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Mate, Vandana
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Knight, Lucy
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Femminella, Grazia Daniela, Frangou, Eleni, Love, Sharon B., Busza, Gail, Holmes, Clive, Ritchie, Craig, Lawrence, Robert, Mcfarlane, Brady, Tadros, George, Ridha, Basil H., Bannister, Carol, Walker, Zuzana, Archer, Hilary, Coulthard, Elizabeth, Underwood, Ben R., Prasanna, Aparna, Koranteng, Paul, Karim, Salman, Junaid, Kehinde, Mcguinness, Bernadette, Nilforooshan, Ramin, Macharouthu, Ajay, Donaldson, Andrew, Thacker, Simon, Russell, Gregor, Malik, Naghma, Mate, Vandana, Knight, Lucy, Kshemendran, Sajeev, Harrison, John, Brooks, David J., Passmore, Anthony Peter, Ballard, Clive and Edison, Paul (2019) Evaluating the effects of the novel GLP-1 analogue liraglutide in Alzheimer’s disease: study protocol for a randomised controlled trial (ELAD study). Trials, 20 (1), [191]. (doi:10.1186/s13063-019-3259-x).

Record type: Article

Abstract

Background
Liraglutide is a glucagon-like peptide-1 (GLP-1) analogue currently approved for type 2 diabetes and obesity. Preclinical evidence in transgenic models of Alzheimer’s disease suggests that liraglutide exerts neuroprotective effects by reducing amyloid oligomers, normalising synaptic plasticity and cerebral glucose uptake, and increasing the proliferation of neuronal progenitor cells. The primary objective of the study is to evaluate the change in cerebral glucose metabolic rate after 12 months of treatment with liraglutide in participants with Alzheimer’s disease compared to those who are receiving placebo.

Methods/design
ELAD is a 12-month, multi-centre, randomised, double-blind, placebo-controlled, phase IIb trial of liraglutide in participants with mild Alzheimer’s dementia. A total of 206 participants will be randomised to receive either liraglutide or placebo as a daily injection for a year. The primary outcome will be the change in cerebral glucose metabolic rate in the cortical regions (hippocampus, medial temporal lobe, and posterior cingulate) from baseline to follow-up in the treatment group compared with the placebo group. The key secondary outcomes are the change from baseline to 12 months in z scores for clinical and cognitive measures (Alzheimer’s Disease Assessment Scale—Cognitive Subscale and Executive domain scores of the Neuropsychological Test Battery, Clinical Dementia Rating Sum of Boxes, and Alzheimer’s Disease Cooperative Study—Activities of Daily Living) and the incidence and severity of treatment-emergent adverse events or clinically important changes in safety assessments. Other secondary outcomes are 12-month change in magnetic resonance imaging volume, diffusion tensor imaging parameters, reduction in microglial activation in a subgroup of participants, reduction in tau formation and change in amyloid levels in a subgroup of participants measured by tau and amyloid imaging, and changes in composite scores using support machine vector analysis in the treatment group compared with the placebo group.

Discussion
Alzheimer’s disease is a leading cause of morbidity worldwide. As available treatments are only symptomatic, the search for disease-modifying therapies is a priority. If the ELAD trial is successful, liraglutide and GLP-1 analogues will represent an important class of compounds to be further evaluated in clinical trials for Alzheimer’s treatment.

Trial registration
ClinicalTrials.gov, NCT01843075. Registration 30 April 2013.

Text
s13063-019-3259-x - Version of Record
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Accepted/In Press date: 27 February 2019
Published date: 3 April 2019

Identifiers

Local EPrints ID: 430645
URI: http://eprints.soton.ac.uk/id/eprint/430645
ISSN: 1745-6215
PURE UUID: 0236c29a-ad50-4a79-80b4-4478d22c83c9
ORCID for Clive Holmes: ORCID iD orcid.org/0000-0003-1999-6912

Catalogue record

Date deposited: 07 May 2019 16:30
Last modified: 16 Mar 2024 03:07

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Contributors

Author: Grazia Daniela Femminella
Author: Eleni Frangou
Author: Sharon B. Love
Author: Gail Busza
Author: Clive Holmes ORCID iD
Author: Craig Ritchie
Author: Robert Lawrence
Author: Brady Mcfarlane
Author: George Tadros
Author: Basil H. Ridha
Author: Carol Bannister
Author: Zuzana Walker
Author: Hilary Archer
Author: Elizabeth Coulthard
Author: Ben R. Underwood
Author: Aparna Prasanna
Author: Paul Koranteng
Author: Salman Karim
Author: Kehinde Junaid
Author: Bernadette Mcguinness
Author: Ramin Nilforooshan
Author: Ajay Macharouthu
Author: Andrew Donaldson
Author: Simon Thacker
Author: Gregor Russell
Author: Naghma Malik
Author: Vandana Mate
Author: Lucy Knight
Author: Sajeev Kshemendran
Author: John Harrison
Author: David J. Brooks
Author: Anthony Peter Passmore
Author: Clive Ballard
Author: Paul Edison

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