The University of Southampton
University of Southampton Institutional Repository

Peri-arterial pathways for clearance of α-Synuclein and tau from the brain: implications for the pathogenesis of dementias and for immunotherapy

Peri-arterial pathways for clearance of α-Synuclein and tau from the brain: implications for the pathogenesis of dementias and for immunotherapy
Peri-arterial pathways for clearance of α-Synuclein and tau from the brain: implications for the pathogenesis of dementias and for immunotherapy

Introduction: Accumulation of amyloid beta (Aβ), α-synuclein (αSyn), and tau in dementias indicates their age-related failure of elimination from the brain. Aβ is eliminated along basement membranes in walls of cerebral arterioles and leptomeningeal arteries (intramural peri-arterial drainage [IPAD]); IPAD is impaired with age. We test the hypothesis that αSyn and tau are also eliminated from the normal brain along IPAD pathways.

Methods: Soluble αSyn or tau was injected into mouse hippocampus. Animals were perfused 5 minutes to 7 days post-injection. Blood vessels were identified by ROX-SE for light-sheet and immunolabeling for confocal microscopy. IPAD was quantified by measuring the proportion of arterioles with αSyn/tau.

Results: αSyn and tau are eliminated from the brain by IPAD but with different dynamics.

Discussion: Age-related failure of IPAD may play a role in the pathogenesis of synucleinopathies and tauopathies. αSyn persists within IPAD at 24 hours, which may affect immunotherapy for αSyn.

2352-8729
Nimmo, Jacqui Taryn
222608e3-256e-4ba4-a259-cbdfcb5340c6
Johnston, David
b41163c9-b9d2-425c-af99-2a357204014e
Dodart, J.C.
7e5f6861-a4e2-4ee7-8d2d-576274e92dc6
MacGregor Sharp, Matthew
ec57c53a-a10a-4b8a-94fe-03eca85ab7c3
Weller, Roy O.
4a501831-e38a-4d39-a125-d7141d6c667b
Nicoll, James
88c0685f-000e-4eb7-8f72-f36b4985e8ed
Verma, Ajay
5a207350-0de9-487a-be28-f9eb2f558140
Carare, Roxana-Octavia
0478c197-b0c1-4206-acae-54e88c8f21fa
Nimmo, Jacqui Taryn
222608e3-256e-4ba4-a259-cbdfcb5340c6
Johnston, David
b41163c9-b9d2-425c-af99-2a357204014e
Dodart, J.C.
7e5f6861-a4e2-4ee7-8d2d-576274e92dc6
MacGregor Sharp, Matthew
ec57c53a-a10a-4b8a-94fe-03eca85ab7c3
Weller, Roy O.
4a501831-e38a-4d39-a125-d7141d6c667b
Nicoll, James
88c0685f-000e-4eb7-8f72-f36b4985e8ed
Verma, Ajay
5a207350-0de9-487a-be28-f9eb2f558140
Carare, Roxana-Octavia
0478c197-b0c1-4206-acae-54e88c8f21fa

Nimmo, Jacqui Taryn, Johnston, David, Dodart, J.C., MacGregor Sharp, Matthew, Weller, Roy O., Nicoll, James, Verma, Ajay and Carare, Roxana-Octavia (2020) Peri-arterial pathways for clearance of α-Synuclein and tau from the brain: implications for the pathogenesis of dementias and for immunotherapy. Alzheimer's & Dementia: Diagnosis, Assessment & Disease Monitoring, 12 (1), [e12070]. (doi:10.1002/dad2.12070).

Record type: Article

Abstract

Introduction: Accumulation of amyloid beta (Aβ), α-synuclein (αSyn), and tau in dementias indicates their age-related failure of elimination from the brain. Aβ is eliminated along basement membranes in walls of cerebral arterioles and leptomeningeal arteries (intramural peri-arterial drainage [IPAD]); IPAD is impaired with age. We test the hypothesis that αSyn and tau are also eliminated from the normal brain along IPAD pathways.

Methods: Soluble αSyn or tau was injected into mouse hippocampus. Animals were perfused 5 minutes to 7 days post-injection. Blood vessels were identified by ROX-SE for light-sheet and immunolabeling for confocal microscopy. IPAD was quantified by measuring the proportion of arterioles with αSyn/tau.

Results: αSyn and tau are eliminated from the brain by IPAD but with different dynamics.

Discussion: Age-related failure of IPAD may play a role in the pathogenesis of synucleinopathies and tauopathies. αSyn persists within IPAD at 24 hours, which may affect immunotherapy for αSyn.

Text
DADM-D-20-00101_R1 - Accepted Manuscript
Download (4MB)

More information

Accepted/In Press date: 22 June 2020
e-pub ahead of print date: 28 July 2020
Published date: 2020
Additional Information: © 2020 The Authors. Alzheimer's & Dementia: Diagnosis, Assessment & Disease Monitoring published by Wiley Periodicals, Inc. on behalf of Alzheimer's Association.

Identifiers

Local EPrints ID: 442059
URI: http://eprints.soton.ac.uk/id/eprint/442059
ISSN: 2352-8729
PURE UUID: 41570b5d-7fee-48d6-9191-a6a8e2e2cfbc
ORCID for David Johnston: ORCID iD orcid.org/0000-0001-6703-6014
ORCID for Matthew MacGregor Sharp: ORCID iD orcid.org/0000-0002-6623-5078
ORCID for James Nicoll: ORCID iD orcid.org/0000-0002-9444-7246
ORCID for Roxana-Octavia Carare: ORCID iD orcid.org/0000-0001-6458-3776

Catalogue record

Date deposited: 06 Jul 2020 16:31
Last modified: 17 Mar 2024 05:41

Export record

Altmetrics

Contributors

Author: Jacqui Taryn Nimmo
Author: David Johnston ORCID iD
Author: J.C. Dodart
Author: Matthew MacGregor Sharp ORCID iD
Author: Roy O. Weller
Author: James Nicoll ORCID iD
Author: Ajay Verma

Download statistics

Downloads from ePrints over the past year. Other digital versions may also be available to download e.g. from the publisher's website.

View more statistics

Atom RSS 1.0 RSS 2.0

Contact ePrints Soton: eprints@soton.ac.uk

ePrints Soton supports OAI 2.0 with a base URL of http://eprints.soton.ac.uk/cgi/oai2

This repository has been built using EPrints software, developed at the University of Southampton, but available to everyone to use.

We use cookies to ensure that we give you the best experience on our website. If you continue without changing your settings, we will assume that you are happy to receive cookies on the University of Southampton website.

×