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Effect of colonisation with Neisseria lactamica on cross-reactive anti-meningococcal B-cell responses: a randomised, controlled, human infection trial

Effect of colonisation with Neisseria lactamica on cross-reactive anti-meningococcal B-cell responses: a randomised, controlled, human infection trial
Effect of colonisation with Neisseria lactamica on cross-reactive anti-meningococcal B-cell responses: a randomised, controlled, human infection trial
Background: Pharyngeal colonisation by the commensal bacterium Neisseria lactamica inhibits colonisation by Neisseria meningitidis and has an inverse epidemiological association with meningococcal disease. The mechanisms that underpin this relationship are unclear, but could involve the induction of cross-reactive immunity. In this study, we aimed to evaluate whether colonisation with N lactamica induces N lactamica-specific B-cell responses that are cross-reactive with N meningitidis.
Methods: In this randomised, placebo-controlled, human infection trial at University Hospital Southampton Clinical Research Facility (Southampton, UK), healthy adults aged 18-45 years were randomly assigned (2:1) to receive intranasal inoculation with either 105 colony-forming units of N lactamica in 1 mL phosphate-buffered saline (PBS) or 1 mL PBS alone. Participants and researchers conducting participant sampling and immunological assays were masked to allocation. The primary endpoint was the frequency of circulating N lactamica-specific plasma cells and memory B cells after N lactamica inoculation (day 7-28) compared with baseline values (day 0), measured using enzyme-linked immunospot assays. The secondary endpoint was to measure the frequency of N meningitidis-specific B cells. In a second study, we measured the effect of duration of N lactamica colonisation on seroconversion by terminating carriage at either 4 days or 14 days with single-dose oral ciprofloxacin. The studies are now closed to participants. The trials are registered with ClinicalTrials.gov, NCT03633474 and NCT03549325.
Findings: Of 50 participants assessed for eligibility between Sept 5, 2018, and March 3, 2019, 31 were randomly assigned (n=20 N lactamica, n=11 PBS). Among the 17 participants who were colonised with N lactamica, the median baselines compared with peak post-colonisation N lactamica-specific plasma-cell frequencies (per 105 peripheral blood mononuclear cells) were 0·0 (IQR 0·0-0·0) versus 5·0 (1·5-10·5) for IgA-secreting plasma cells (p<0·0001), and 0·0 (0·0-0·0) versus 3·0 (1·5-9·5) for IgG-secreting plasma cells (p<0·0001). Median N lactamica-specific IgG memory-B-cell frequencies (percentage of total IgG memory B cells) increased from 0·0024% (0·0000-0·0097) at baseline to 0·0384% (0·0275-0·0649) at day 28 (p<0·0001). The frequency of N meningitidis-specific IgA-secreting and IgG-secreting plasma cells and memory B cells also increased signficantly in participants who were colonised with N lactamica. Upper respiratory tract symptoms were reported in ten (50%) of 20 participants who were inoculated with N lactamica and six (55%) of 11 participants who were inoculated with PBS (p>0·99). Three additional adverse events (two in the N lactamica group and one in the PBS group) and no serious adverse events were reported. In the second study, anti-N lactamica and anti-N meningitidis serum IgG titres increased only in participants who were colonised with N lactamica for 14 days.
Interpretation: Natural immunity to N meningitidis after colonisation with N lactamica might be due to cross-reactive adaptive responses. Exploitation of this microbial mechanism with a genetically modified live vector could protect against N meningitidis colonisation and disease.
Funding: Wellcome Trust, Medical Research Council, and NIHR Southampton Biomedical Research Centre.
e931-e943
Dale, Adam P
5096a630-1d0b-4e37-a1d4-e971e08acb54
Theodosiou, Anastasia A
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Gbesemete, Diane F
2543380e-16b0-42b4-9d0f-88421ac33106
Guy, Jonathan M
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Jones, Eleanor F
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Hill, Alison R
de7a9d4f-7c5c-440c-9619-5b390f6347fd
Ibrahim, Muktar M
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de Graaf, Hans
447e78ed-346f-45bb-9238-fce2118d5559
Ahmed, Muhammad
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Faust, Saul N
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Gorringe, Andrew R
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Polak, Marta E
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Laver, Jay R
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Read, Robert C
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Dale, Adam P
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Theodosiou, Anastasia A
c6e63581-c22d-4a2c-9d14-2e66594eb053
Gbesemete, Diane F
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Guy, Jonathan M
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Jones, Eleanor F
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Hill, Alison R
de7a9d4f-7c5c-440c-9619-5b390f6347fd
Ibrahim, Muktar M
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de Graaf, Hans
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Ahmed, Muhammad
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Faust, Saul N
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Gorringe, Andrew R
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Polak, Marta E
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Laver, Jay R
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Read, Robert C
b5caca7b-0063-438a-b703-7ecbb6fc2b51

Dale, Adam P, Theodosiou, Anastasia A, Gbesemete, Diane F, Guy, Jonathan M, Jones, Eleanor F, Hill, Alison R, Ibrahim, Muktar M, de Graaf, Hans, Ahmed, Muhammad, Faust, Saul N, Gorringe, Andrew R, Polak, Marta E, Laver, Jay R and Read, Robert C (2022) Effect of colonisation with Neisseria lactamica on cross-reactive anti-meningococcal B-cell responses: a randomised, controlled, human infection trial. The Lancet Microbe, 3 (12), e931-e943. (doi:10.1016/S2666-5247(22)00283-X).

Record type: Article

Abstract

Background: Pharyngeal colonisation by the commensal bacterium Neisseria lactamica inhibits colonisation by Neisseria meningitidis and has an inverse epidemiological association with meningococcal disease. The mechanisms that underpin this relationship are unclear, but could involve the induction of cross-reactive immunity. In this study, we aimed to evaluate whether colonisation with N lactamica induces N lactamica-specific B-cell responses that are cross-reactive with N meningitidis.
Methods: In this randomised, placebo-controlled, human infection trial at University Hospital Southampton Clinical Research Facility (Southampton, UK), healthy adults aged 18-45 years were randomly assigned (2:1) to receive intranasal inoculation with either 105 colony-forming units of N lactamica in 1 mL phosphate-buffered saline (PBS) or 1 mL PBS alone. Participants and researchers conducting participant sampling and immunological assays were masked to allocation. The primary endpoint was the frequency of circulating N lactamica-specific plasma cells and memory B cells after N lactamica inoculation (day 7-28) compared with baseline values (day 0), measured using enzyme-linked immunospot assays. The secondary endpoint was to measure the frequency of N meningitidis-specific B cells. In a second study, we measured the effect of duration of N lactamica colonisation on seroconversion by terminating carriage at either 4 days or 14 days with single-dose oral ciprofloxacin. The studies are now closed to participants. The trials are registered with ClinicalTrials.gov, NCT03633474 and NCT03549325.
Findings: Of 50 participants assessed for eligibility between Sept 5, 2018, and March 3, 2019, 31 were randomly assigned (n=20 N lactamica, n=11 PBS). Among the 17 participants who were colonised with N lactamica, the median baselines compared with peak post-colonisation N lactamica-specific plasma-cell frequencies (per 105 peripheral blood mononuclear cells) were 0·0 (IQR 0·0-0·0) versus 5·0 (1·5-10·5) for IgA-secreting plasma cells (p<0·0001), and 0·0 (0·0-0·0) versus 3·0 (1·5-9·5) for IgG-secreting plasma cells (p<0·0001). Median N lactamica-specific IgG memory-B-cell frequencies (percentage of total IgG memory B cells) increased from 0·0024% (0·0000-0·0097) at baseline to 0·0384% (0·0275-0·0649) at day 28 (p<0·0001). The frequency of N meningitidis-specific IgA-secreting and IgG-secreting plasma cells and memory B cells also increased signficantly in participants who were colonised with N lactamica. Upper respiratory tract symptoms were reported in ten (50%) of 20 participants who were inoculated with N lactamica and six (55%) of 11 participants who were inoculated with PBS (p>0·99). Three additional adverse events (two in the N lactamica group and one in the PBS group) and no serious adverse events were reported. In the second study, anti-N lactamica and anti-N meningitidis serum IgG titres increased only in participants who were colonised with N lactamica for 14 days.
Interpretation: Natural immunity to N meningitidis after colonisation with N lactamica might be due to cross-reactive adaptive responses. Exploitation of this microbial mechanism with a genetically modified live vector could protect against N meningitidis colonisation and disease.
Funding: Wellcome Trust, Medical Research Council, and NIHR Southampton Biomedical Research Centre.

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e-pub ahead of print date: 30 November 2022
Published date: 30 November 2022
Additional Information: Funding: Wellcome Trust, Medical Research Council, and NIHR Southampton Biomedical Research Centre. This work was funded by a: Wellcome Trust Research Training Fellowship (203581/Z/16/Z to APD); and by Medical Research Council grants (MR/N013204/1 and MR/N026993/1 to RCR). RCR (NF­SI­0617–10010) and SNF (NIHR 201409) are NIHR Senior Investigators. This work was also supported by the NIHR Southampton Biomedical Research Centre (IS­BRC­1215–20004) and the NIHR Southampton AMR Clinical Research Laboratory (NIHR 200638).

Identifiers

Local EPrints ID: 472898
URI: http://eprints.soton.ac.uk/id/eprint/472898
PURE UUID: 4f42d462-d923-4f41-8d52-4d7b29640582
ORCID for Adam P Dale: ORCID iD orcid.org/0000-0001-8163-7481
ORCID for Alison R Hill: ORCID iD orcid.org/0000-0001-5397-873X
ORCID for Muhammad Ahmed: ORCID iD orcid.org/0000-0003-3234-2788
ORCID for Saul N Faust: ORCID iD orcid.org/0000-0003-3410-7642
ORCID for Jay R Laver: ORCID iD orcid.org/0000-0003-3314-5989
ORCID for Robert C Read: ORCID iD orcid.org/0000-0002-4297-6728

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Date deposited: 05 Jan 2023 18:01
Last modified: 04 Sep 2024 01:52

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Contributors

Author: Adam P Dale ORCID iD
Author: Anastasia A Theodosiou
Author: Diane F Gbesemete
Author: Jonathan M Guy
Author: Eleanor F Jones
Author: Alison R Hill ORCID iD
Author: Muktar M Ibrahim
Author: Hans de Graaf
Author: Muhammad Ahmed ORCID iD
Author: Saul N Faust ORCID iD
Author: Andrew R Gorringe
Author: Marta E Polak
Author: Jay R Laver ORCID iD
Author: Robert C Read ORCID iD

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