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Analyses of 1,236 genotyped primary ciliary dyskinesia individuals identify regional clusters of distinct DNA variants and significant genotype-phenotype correlations

Analyses of 1,236 genotyped primary ciliary dyskinesia individuals identify regional clusters of distinct DNA variants and significant genotype-phenotype correlations
Analyses of 1,236 genotyped primary ciliary dyskinesia individuals identify regional clusters of distinct DNA variants and significant genotype-phenotype correlations
Background: primary ciliary dyskinesia (PCD) represents a group of rare hereditary disorders characterized by deficient ciliary airway clearance that can be associated with laterality defects. We aimed to describe the underlying gene defects, geographical differences in genotypes and their relationship to diagnostic findings and clinical phenotypes.

Methods: genetic variants and clinical findings (age, sex, body mass index, laterality defects, FEV1) were collected from 19 countries using the ERN LUNG International PCD Registry. Genetic data were evaluated according to ACMG guidelines. We assessed regional distribution of implicated genes and genetic variants as well as genotype correlations with laterality defects and FEV1.

Results: 1236 individuals carried 908 distinct pathogenic DNA variants in 46 PCD genes. We found considerable variation in the distribution of PCD genotypes across countries due to the presence of distinct founder variants. The prevalence of PCD genotypes associated with pathognomonic ultrastructural defects (mean 72%; 47–100%) and laterality defects (mean 42%; 28–69%) varied widely among the countries. The prevalence of laterality defects was significantly lower in PCD individuals without pathognomonic ciliary ultrastructure defects (18%). The PCD cohort had a reduced median FEV1 z-score (−1.66). In the group of individuals with CCNO (−3.26), CCDC39 (−2.49), and CCDC40 (−2.96) variants, FEV1 z-scores were significantly lower, while the group of DNAH11 (−0.83) and ODAD1 (−0.85) variant individuals had significantly milder FEV1 z-score reductions compared to the whole PCD cohort.

Conclusion: this unprecedented multinational dataset of DNA variants and information on their distribution across countries facilitates interpretation of genetic epidemiology of PCD and provides prediction of diagnostic and phenotypic features such as the course of lung function.
0903-1936
Raidt, Johanna
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Riepenhausen, Sarah
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Pennekamp, Petra
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Olbrich, Heike
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Amirav, Israel
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Athanazio, Rodrigo Abensur
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Aviram, M.
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Balinotti, J.
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Bar-on, O.
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Lucas, Jane
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Walker, Woolf
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Wetzke, M.
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Witt, M.
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Yiallouros, Panayiotis
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Zschocke, A.
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Ziętkiewicz, E.
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Nielsen, Kim G.
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Omran, Heymut
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et al.
Raidt, Johanna
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Riepenhausen, Sarah
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Pennekamp, Petra
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Olbrich, Heike
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Amirav, Israel
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Athanazio, Rodrigo Abensur
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Aviram, M.
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Balinotti, J.
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Bar-on, O.
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Lucas, Jane
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Walker, Woolf
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Wetzke, M.
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Witt, M.
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Yiallouros, Panayiotis
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Zschocke, A.
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Ziętkiewicz, E.
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Nielsen, Kim G.
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Omran, Heymut
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Raidt, Johanna, Riepenhausen, Sarah and Pennekamp, Petra , et al. (2024) Analyses of 1,236 genotyped primary ciliary dyskinesia individuals identify regional clusters of distinct DNA variants and significant genotype-phenotype correlations. European Respiratory Journal, 63 (6). (doi:10.1183/13993003.01769-2023).

Record type: Article

Abstract

Background: primary ciliary dyskinesia (PCD) represents a group of rare hereditary disorders characterized by deficient ciliary airway clearance that can be associated with laterality defects. We aimed to describe the underlying gene defects, geographical differences in genotypes and their relationship to diagnostic findings and clinical phenotypes.

Methods: genetic variants and clinical findings (age, sex, body mass index, laterality defects, FEV1) were collected from 19 countries using the ERN LUNG International PCD Registry. Genetic data were evaluated according to ACMG guidelines. We assessed regional distribution of implicated genes and genetic variants as well as genotype correlations with laterality defects and FEV1.

Results: 1236 individuals carried 908 distinct pathogenic DNA variants in 46 PCD genes. We found considerable variation in the distribution of PCD genotypes across countries due to the presence of distinct founder variants. The prevalence of PCD genotypes associated with pathognomonic ultrastructural defects (mean 72%; 47–100%) and laterality defects (mean 42%; 28–69%) varied widely among the countries. The prevalence of laterality defects was significantly lower in PCD individuals without pathognomonic ciliary ultrastructure defects (18%). The PCD cohort had a reduced median FEV1 z-score (−1.66). In the group of individuals with CCNO (−3.26), CCDC39 (−2.49), and CCDC40 (−2.96) variants, FEV1 z-scores were significantly lower, while the group of DNAH11 (−0.83) and ODAD1 (−0.85) variant individuals had significantly milder FEV1 z-score reductions compared to the whole PCD cohort.

Conclusion: this unprecedented multinational dataset of DNA variants and information on their distribution across countries facilitates interpretation of genetic epidemiology of PCD and provides prediction of diagnostic and phenotypic features such as the course of lung function.

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Genotypes_PCD (1) - Accepted Manuscript
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More information

Accepted/In Press date: 3 May 2024
e-pub ahead of print date: 13 June 2024
Published date: 13 June 2024

Identifiers

Local EPrints ID: 491856
URI: http://eprints.soton.ac.uk/id/eprint/491856
ISSN: 0903-1936
PURE UUID: 995e258b-4411-45c6-b9e8-1cb71c45985f
ORCID for Jane Lucas: ORCID iD orcid.org/0000-0001-8701-9975

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Date deposited: 04 Jul 2024 17:19
Last modified: 12 Jul 2024 01:40

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Contributors

Author: Johanna Raidt
Author: Sarah Riepenhausen
Author: Petra Pennekamp
Author: Heike Olbrich
Author: Israel Amirav
Author: Rodrigo Abensur Athanazio
Author: M. Aviram
Author: J. Balinotti
Author: O. Bar-on
Author: Jane Lucas ORCID iD
Author: Woolf Walker
Author: M. Wetzke
Author: M. Witt
Author: Panayiotis Yiallouros
Author: A. Zschocke
Author: E. Ziętkiewicz
Author: Kim G. Nielsen
Author: Heymut Omran
Corporate Author: et al.

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