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Shared genetic variants suggest common pathways in allergy and autoimmune diseases

Shared genetic variants suggest common pathways in allergy and autoimmune diseases
Shared genetic variants suggest common pathways in allergy and autoimmune diseases
Background
The relationship between allergy and autoimmune disorders is complex and poorly understood.

Objective
We sought to investigate commonalities in genetic loci and pathways between allergy and autoimmune diseases to elucidate shared disease mechanisms.

Methods
We meta-analyzed 2 genome-wide association studies on self-reported allergy and sensitization comprising a total of 62,330 subjects. These results were used to calculate enrichment for single nucleotide polymorphisms (SNPs) previously associated with autoimmune diseases. Furthermore, we probed for enrichment within genetic pathways and of transcription factor binding sites and characterized commonalities in variant burden on tissue-specific regulatory sites by calculating the enrichment of allergy SNPs falling in gene regulatory regions in various cells using Encode Roadmap DNase-hypersensitive site data. Finally, we compared the allergy data with those of all known diseases.

Results
Among 290 loci previously associated with 16 autoimmune diseases, we found a significant enrichment of loci also associated with allergy (P = 1.4e-17) encompassing 29 loci at a false discovery rate of less than 0.05. Such enrichment seemed to be a general characteristic for autoimmune diseases. Among the common loci, 48% had the same direction of effect for allergy and autoimmune diseases. Additionally, we observed an enrichment of allergy SNPs falling within immune pathways and regions of chromatin accessible in immune cells that was also represented in patients with autoimmune diseases but not those with other diseases.

Conclusion
We identified shared susceptibility loci and commonalities in pathways between allergy and autoimmune diseases, suggesting shared disease mechanisms. Further studies of these shared genetic mechanisms might help in understanding the complex relationship between these diseases, including the parallel increase in disease prevalence.
0091-6749
771 - 781
Kreiner, E.
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Waage, J.
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Standl, M.
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Brix, S.
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Pers, T.H.
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Couto Alves, A.
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Warrington, N.M.
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Tiesler, C.M.T.
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Fuertes, E.
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Franke, L.
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Hirschhorn, J.N.
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James, A.
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Simpson, A.
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Tung, J.Y.
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Koppelman, G.H.
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Postma, D.S.
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Pennell, C.E.
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Jarvelin, M.-R.
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Custovic, A.
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Timpson, N.
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Ferreira, M.A.
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Strachan, D.P.
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Henderson, J.
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Hinds, D.
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Bisgaard, H.
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Bønnelykke, K.
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Kreiner, E.
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Waage, J.
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Standl, M.
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Brix, S.
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Pers, T.H.
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Couto Alves, A.
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Warrington, N.M.
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Tiesler, C.M.T.
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Fuertes, E.
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Franke, L.
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Hirschhorn, J.N.
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James, A.
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Simpson, A.
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Tung, J.Y.
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Koppelman, G.H.
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Postma, D.S.
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Pennell, C.E.
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Jarvelin, M.-R.
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Custovic, A.
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Timpson, N.
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Ferreira, M.A.
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Strachan, D.P.
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Henderson, J.
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Hinds, D.
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Bisgaard, H.
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Bønnelykke, K.
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Kreiner, E., Waage, J., Standl, M., Brix, S., Pers, T.H., Couto Alves, A., Warrington, N.M., Tiesler, C.M.T., Fuertes, E., Franke, L., Hirschhorn, J.N., James, A., Simpson, A., Tung, J.Y., Koppelman, G.H., Postma, D.S., Pennell, C.E., Jarvelin, M.-R., Custovic, A., Timpson, N., Ferreira, M.A., Strachan, D.P., Henderson, J., Hinds, D., Bisgaard, H. and Bønnelykke, K. (2017) Shared genetic variants suggest common pathways in allergy and autoimmune diseases. Journal of Allergy and Clinical Immunology, 140 (3), 771 - 781. (doi:10.1016/j.jaci.2016.10.055).

Record type: Article

Abstract

Background
The relationship between allergy and autoimmune disorders is complex and poorly understood.

Objective
We sought to investigate commonalities in genetic loci and pathways between allergy and autoimmune diseases to elucidate shared disease mechanisms.

Methods
We meta-analyzed 2 genome-wide association studies on self-reported allergy and sensitization comprising a total of 62,330 subjects. These results were used to calculate enrichment for single nucleotide polymorphisms (SNPs) previously associated with autoimmune diseases. Furthermore, we probed for enrichment within genetic pathways and of transcription factor binding sites and characterized commonalities in variant burden on tissue-specific regulatory sites by calculating the enrichment of allergy SNPs falling in gene regulatory regions in various cells using Encode Roadmap DNase-hypersensitive site data. Finally, we compared the allergy data with those of all known diseases.

Results
Among 290 loci previously associated with 16 autoimmune diseases, we found a significant enrichment of loci also associated with allergy (P = 1.4e-17) encompassing 29 loci at a false discovery rate of less than 0.05. Such enrichment seemed to be a general characteristic for autoimmune diseases. Among the common loci, 48% had the same direction of effect for allergy and autoimmune diseases. Additionally, we observed an enrichment of allergy SNPs falling within immune pathways and regions of chromatin accessible in immune cells that was also represented in patients with autoimmune diseases but not those with other diseases.

Conclusion
We identified shared susceptibility loci and commonalities in pathways between allergy and autoimmune diseases, suggesting shared disease mechanisms. Further studies of these shared genetic mechanisms might help in understanding the complex relationship between these diseases, including the parallel increase in disease prevalence.

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More information

Accepted/In Press date: 11 October 2016
Published date: 17 February 2017

Identifiers

Local EPrints ID: 494998
URI: http://eprints.soton.ac.uk/id/eprint/494998
ISSN: 0091-6749
PURE UUID: 4622ac53-c0bc-4e3c-933e-7e3270ea88b1
ORCID for A. Couto Alves: ORCID iD orcid.org/0000-0001-8519-7356
ORCID for M.A. Ferreira: ORCID iD orcid.org/0000-0002-2428-0284

Catalogue record

Date deposited: 25 Oct 2024 16:34
Last modified: 26 Oct 2024 02:12

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Contributors

Author: E. Kreiner
Author: J. Waage
Author: M. Standl
Author: S. Brix
Author: T.H. Pers
Author: A. Couto Alves ORCID iD
Author: N.M. Warrington
Author: C.M.T. Tiesler
Author: E. Fuertes
Author: L. Franke
Author: J.N. Hirschhorn
Author: A. James
Author: A. Simpson
Author: J.Y. Tung
Author: G.H. Koppelman
Author: D.S. Postma
Author: C.E. Pennell
Author: M.-R. Jarvelin
Author: A. Custovic
Author: N. Timpson
Author: M.A. Ferreira ORCID iD
Author: D.P. Strachan
Author: J. Henderson
Author: D. Hinds
Author: H. Bisgaard
Author: K. Bønnelykke

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