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Cytogenetic and molecular monitoring of residual disease in chronic myeloid leukaemia

Cytogenetic and molecular monitoring of residual disease in chronic myeloid leukaemia
Cytogenetic and molecular monitoring of residual disease in chronic myeloid leukaemia
For patients with chronic myeloid leukaemia, methods for monitoring response to treatment have changed considerably in recent years. In the 1980s, the principal approach was repeated examination of bone marrow metaphases for the presence of the Ph chromosome in patients treated by interferon-alpha (IFN-alpha) or allogeneic stem cell transplantation. The use of fluorescence in situ hybridisation (FISH) techniques to detect the BCR-ABL fusion gene in Ph-positive leukaemia cells increased the sensitivity of cytogenetic studies to some degree. In the last 10 years, the reverse-transcriptase polymerase chain reaction (RT-PCR) has proved extremely valuable for assessing and monitoring minimal residual disease in patients who achieve Ph negativity after treatment with IFN-alpha or with the new Abl tyrosine kinase inhibitor imatinib mesylate or after allogeneic stem cell transplantation (SCT). Results are consistent with the notion that the majority of long-term survivors after allogeneic SCT are probably 'cured'; for other patients monitored serially in complete cytogenetic remission, rising numbers of BCR-ABL transcripts detected by RT-PCR can indicate the need for further therapy
neoplasm proteins, stem cell transplantation, interferon-alpha, methods, review, rna, standards, therapy, bone, proteins, southern, london, western, neoplasm, bone marrow, residual, pathology, humans, cytogenetic analysis, polymerase chain reaction, protein, myeloid, analysis, cell transplantation, leukemia, fluorescence, transplantation, blotting, metaphase, diagnosis, tyrosine, chronic, patients, disease, reverse transcriptase polymerase chain reaction, genetics
0001-5792
64-75
Kaeda, J.
97eba745-9c30-4333-9235-00c915b12cf6
Chase, A.
a40a09c2-3073-4655-ba0b-a802e34914b5
Goldman, J.M.
c0abeb85-851b-4680-b28e-e646d94a91f7
Kaeda, J.
97eba745-9c30-4333-9235-00c915b12cf6
Chase, A.
a40a09c2-3073-4655-ba0b-a802e34914b5
Goldman, J.M.
c0abeb85-851b-4680-b28e-e646d94a91f7

Kaeda, J., Chase, A. and Goldman, J.M. (2002) Cytogenetic and molecular monitoring of residual disease in chronic myeloid leukaemia. Acta Haematologica, 107 (2), 64-75. (doi:10.1159/000068096).

Record type: Article

Abstract

For patients with chronic myeloid leukaemia, methods for monitoring response to treatment have changed considerably in recent years. In the 1980s, the principal approach was repeated examination of bone marrow metaphases for the presence of the Ph chromosome in patients treated by interferon-alpha (IFN-alpha) or allogeneic stem cell transplantation. The use of fluorescence in situ hybridisation (FISH) techniques to detect the BCR-ABL fusion gene in Ph-positive leukaemia cells increased the sensitivity of cytogenetic studies to some degree. In the last 10 years, the reverse-transcriptase polymerase chain reaction (RT-PCR) has proved extremely valuable for assessing and monitoring minimal residual disease in patients who achieve Ph negativity after treatment with IFN-alpha or with the new Abl tyrosine kinase inhibitor imatinib mesylate or after allogeneic stem cell transplantation (SCT). Results are consistent with the notion that the majority of long-term survivors after allogeneic SCT are probably 'cured'; for other patients monitored serially in complete cytogenetic remission, rising numbers of BCR-ABL transcripts detected by RT-PCR can indicate the need for further therapy

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More information

Published date: 2002
Keywords: neoplasm proteins, stem cell transplantation, interferon-alpha, methods, review, rna, standards, therapy, bone, proteins, southern, london, western, neoplasm, bone marrow, residual, pathology, humans, cytogenetic analysis, polymerase chain reaction, protein, myeloid, analysis, cell transplantation, leukemia, fluorescence, transplantation, blotting, metaphase, diagnosis, tyrosine, chronic, patients, disease, reverse transcriptase polymerase chain reaction, genetics

Identifiers

Local EPrints ID: 59901
URI: http://eprints.soton.ac.uk/id/eprint/59901
ISSN: 0001-5792
PURE UUID: ec8f175d-0af4-4481-9fb2-54a89f0124f6
ORCID for A. Chase: ORCID iD orcid.org/0000-0001-6617-9953

Catalogue record

Date deposited: 02 Sep 2008
Last modified: 15 Mar 2024 11:18

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Contributors

Author: J. Kaeda
Author: A. Chase ORCID iD
Author: J.M. Goldman

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