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Childhood growth and hypertension in later life

Childhood growth and hypertension in later life
Childhood growth and hypertension in later life
Few studies have examined the effects of both prenatal and postnatal growth on hypertension. We report on hypertension in 2003 people aged 62 years who were randomly selected from the Helsinki birth cohort and examined in a clinic. Their heights and weights had been recorded serially up to age 11 years. A total of 644 had already been diagnosed with hypertension. Compared with normotensive people, they were obese and insulin resistant. At birth they were thin and short, and they gained weight slowly up to age 2 years; thereafter they grew rapidly so that at age 11 years their body size was around the average. The odds ratio associated with each kilogram of birthweight was 0.42 (95% CI: 0.32 to 0.56); with each 10 kg of current weight it was 1.85 (95% CI: 1.66 to 2.05). The blood pressures of another 802 people were classified as hypertensive under current definitions. They were overweight and had an atherogenic lipid profile. At birth they were short, and after birth they grew slowly so that at age 11 years they were short and thin. The odds ratio associated with each kilogram of weight at age 2 years was 0.75 (95% CI: 0.68 to 0.84); with each 10 kg of current weight it was 1.42 (95% CI: 1.28 to 1.57). We conclude that 2 different paths of childhood growth precede the development of hypertension. We suggest that they lead to hypertension through different biological mechanisms and may respond differently to medication.
body weight, cohort studies, physiopathology, birth weight, diabetes, weight, etiology, body size, chronic disease, lipids, development, physiology, health, birth, blood, research, disease, childhood, male, coronary disease, aging, glucose, stroke, odds ratio, insulin, Finland, size, growth, middle aged, public health, overweight, hypertension, blood pressure, height, humans, body height, metabolism, aged, cohort, report, human development, blood glucose, female
0194-911X
1415-1421
Eriksson, Johan G.
eb96b1c5-af07-4a52-8a73-7541451d32cd
Forsen, Tom J.
55342e20-c4a5-40e5-ad81-2a64c1e0f333
Kajantie, Eero
d68d55b6-6df1-4195-a914-44c738a6db93
Osmond, Clive
2677bf85-494f-4a78-adf8-580e1b8acb81
Barker, David J.
1c926291-c5bd-4f53-8827-6ae435fa97ea
Eriksson, Johan G.
eb96b1c5-af07-4a52-8a73-7541451d32cd
Forsen, Tom J.
55342e20-c4a5-40e5-ad81-2a64c1e0f333
Kajantie, Eero
d68d55b6-6df1-4195-a914-44c738a6db93
Osmond, Clive
2677bf85-494f-4a78-adf8-580e1b8acb81
Barker, David J.
1c926291-c5bd-4f53-8827-6ae435fa97ea

Eriksson, Johan G., Forsen, Tom J., Kajantie, Eero, Osmond, Clive and Barker, David J. (2007) Childhood growth and hypertension in later life. Hypertension, 49 (6), 1415-1421. (doi:10.1161/HYPERTENSIONAHA.106.085597).

Record type: Article

Abstract

Few studies have examined the effects of both prenatal and postnatal growth on hypertension. We report on hypertension in 2003 people aged 62 years who were randomly selected from the Helsinki birth cohort and examined in a clinic. Their heights and weights had been recorded serially up to age 11 years. A total of 644 had already been diagnosed with hypertension. Compared with normotensive people, they were obese and insulin resistant. At birth they were thin and short, and they gained weight slowly up to age 2 years; thereafter they grew rapidly so that at age 11 years their body size was around the average. The odds ratio associated with each kilogram of birthweight was 0.42 (95% CI: 0.32 to 0.56); with each 10 kg of current weight it was 1.85 (95% CI: 1.66 to 2.05). The blood pressures of another 802 people were classified as hypertensive under current definitions. They were overweight and had an atherogenic lipid profile. At birth they were short, and after birth they grew slowly so that at age 11 years they were short and thin. The odds ratio associated with each kilogram of weight at age 2 years was 0.75 (95% CI: 0.68 to 0.84); with each 10 kg of current weight it was 1.42 (95% CI: 1.28 to 1.57). We conclude that 2 different paths of childhood growth precede the development of hypertension. We suggest that they lead to hypertension through different biological mechanisms and may respond differently to medication.

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More information

Published date: 2007
Keywords: body weight, cohort studies, physiopathology, birth weight, diabetes, weight, etiology, body size, chronic disease, lipids, development, physiology, health, birth, blood, research, disease, childhood, male, coronary disease, aging, glucose, stroke, odds ratio, insulin, Finland, size, growth, middle aged, public health, overweight, hypertension, blood pressure, height, humans, body height, metabolism, aged, cohort, report, human development, blood glucose, female

Identifiers

Local EPrints ID: 61099
URI: http://eprints.soton.ac.uk/id/eprint/61099
ISSN: 0194-911X
PURE UUID: 0bbd7411-6dcb-4d97-bc80-158663bc8982
ORCID for Clive Osmond: ORCID iD orcid.org/0000-0002-9054-4655

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Date deposited: 12 Sep 2008
Last modified: 16 Mar 2024 02:50

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Contributors

Author: Johan G. Eriksson
Author: Tom J. Forsen
Author: Eero Kajantie
Author: Clive Osmond ORCID iD
Author: David J. Barker

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