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Birth weight, stress, and the metabolic syndrome in adult life

Birth weight, stress, and the metabolic syndrome in adult life
Birth weight, stress, and the metabolic syndrome in adult life
There is now widespread agreement that small size at birth is associated with an increased risk of the metabolic syndrome (glucose intolerance, high blood pressure, and dyslipidemia) and related pathologies, including cardiovascular disease in later life. Evidence is emerging that suggests that programming of hormonal systems in response to an adverse fetal environment may be one of the mechanisms underlying these long-term consequences of growth restriction in early life. In particular, alterations in neuroendocrine responses to stress may be important. Recent research suggests that increased adrenocortical and sympathoadrenal responses are associated with small size at birth. Epidemiological studies show that such physiological alterations in these neuroendocrine systems may have potent effects on risk of cardiovascular disease through their influence on risk factors, such as plasma glucose and lipid concentrations and blood pressure
size, cardiovascular-disease, adult, blood, birth weight, syndrome, early-life, fetal, disease, risk factors, birth, responses, consequences, risk, research, programming, pressure, birth-weight, plasma, environment, blood pressure, stress, weight, metabolic syndrome, cardiovascular disease, growth, pathology, glucose intolerance, cardiovascular, later life, glucose, blood-pressure
0077-8923
28-36
Phillips, D.I.
29b73be7-2ff9-4fff-ae42-d59842df4cc6
Jones, A.
bcae84a4-4191-4a3e-b695-1b6e2b0681c7
Goulden, P.A.
8def8779-641f-409f-ba5f-469407bfb375
Phillips, D.I.
29b73be7-2ff9-4fff-ae42-d59842df4cc6
Jones, A.
bcae84a4-4191-4a3e-b695-1b6e2b0681c7
Goulden, P.A.
8def8779-641f-409f-ba5f-469407bfb375

Phillips, D.I., Jones, A. and Goulden, P.A. (2006) Birth weight, stress, and the metabolic syndrome in adult life. Annals of the New York Academy of Sciences, 1083, 28-36.

Record type: Article

Abstract

There is now widespread agreement that small size at birth is associated with an increased risk of the metabolic syndrome (glucose intolerance, high blood pressure, and dyslipidemia) and related pathologies, including cardiovascular disease in later life. Evidence is emerging that suggests that programming of hormonal systems in response to an adverse fetal environment may be one of the mechanisms underlying these long-term consequences of growth restriction in early life. In particular, alterations in neuroendocrine responses to stress may be important. Recent research suggests that increased adrenocortical and sympathoadrenal responses are associated with small size at birth. Epidemiological studies show that such physiological alterations in these neuroendocrine systems may have potent effects on risk of cardiovascular disease through their influence on risk factors, such as plasma glucose and lipid concentrations and blood pressure

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More information

Published date: 2006
Keywords: size, cardiovascular-disease, adult, blood, birth weight, syndrome, early-life, fetal, disease, risk factors, birth, responses, consequences, risk, research, programming, pressure, birth-weight, plasma, environment, blood pressure, stress, weight, metabolic syndrome, cardiovascular disease, growth, pathology, glucose intolerance, cardiovascular, later life, glucose, blood-pressure

Identifiers

Local EPrints ID: 61432
URI: http://eprints.soton.ac.uk/id/eprint/61432
ISSN: 0077-8923
PURE UUID: 9bf60c76-8416-4b6d-ac48-0ec74a1e4b7b

Catalogue record

Date deposited: 03 Sep 2008
Last modified: 22 Jul 2022 21:14

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Contributors

Author: D.I. Phillips
Author: A. Jones
Author: P.A. Goulden

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