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t(6;14)(p22;q32): a new recurrent IGH@ translocation involving ID4 in B-cell precursor acute lymphoblastic leukemia (BCP-ALL)

t(6;14)(p22;q32): a new recurrent IGH@ translocation involving ID4 in B-cell precursor acute lymphoblastic leukemia (BCP-ALL)
t(6;14)(p22;q32): a new recurrent IGH@ translocation involving ID4 in B-cell precursor acute lymphoblastic leukemia (BCP-ALL)
Translocations involving the immunoglobulin heavy chain locus (IGH@) at chromosome band 14q32 are common in mature B-cell neoplasms, but are rare in B-cell precursor acute lymphoblastic leukemia (BCP-ALL). Here, we report the translocation, t(6;14)(p22;q32), involving IGH@ as a novel recurrent translocation in 13 BCP-ALL patients. Fluorescence in situ hybridization and long-distance inverse polymerase chain reaction (PCR) identified ID4 as the partner gene. Breakpoints were scattered over a 19kb region centromeric of ID4. Quantitative real-time PCR showed up-regulation of ID4 mRNA. All patients had deletions of CDKN2A and PAX5 located on the short arm of chromosome 9, frequently as a result of an isochromosome, i(9)(q10) (9/13, 69%). This study defines a new subgroup of BCP-ALL characterized by ID4 over-expression and CDKN2A and PAX5 deletions. Preliminary survival data suggest that this subgroup may be associated with a good response to therapy.
apoptosis, germany, neoplasms, human, leukemia, activation, expression, malignancies, overexpression, t(14, loop-helix proteins, survival, time, cancer, london, deletions, blood, in situ hybridization, patients, gene, report, fluorescence, england, lymphocytic-leukemia, arm, therapy, polymerase chain reaction, hematology, 19)(q32, q13), up-regulation
0006-4971
387-391
Russell, L.J.
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Akasaka, T.
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Majid, A.
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Sugimoto, K.J.
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Karran, E.L.
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Nagel, I.
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Harder, L.
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Claviez, A.
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Gesk, S.
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Moorman, A.V.
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Ross, F.
ec0958f8-b992-4e4a-b7e3-c474600390ba
Mazzullo, H.
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Strefford, J.C.
3782b392-f080-42bf-bdca-8aa5d6ca532f
Siebert, R.
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Dyer, M.J.S.
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Harrison, C.J.
9c9f6b47-8bfc-49dd-b156-74539b170291
Russell, L.J.
7e3f016b-6299-4e23-ab6c-8a6afdddbb44
Akasaka, T.
2ea30760-d6ce-4836-a73a-caacb7fde7af
Majid, A.
8c2bf857-5d12-4016-90d1-aacf9d1c89d3
Sugimoto, K.J.
137f3ff3-999d-46fc-88b9-915d15d42d29
Karran, E.L.
0abb1b2f-fe06-481a-a023-aa11054f315c
Nagel, I.
9882d290-7595-4ffa-81a3-117fb1d054a1
Harder, L.
925f33a9-c986-4e6f-ac9c-e0071af89b08
Claviez, A.
6f4887db-f601-4e8b-9865-ac6b24812a9e
Gesk, S.
4bdb8b0c-6666-4d1e-8c40-f9ce85686c34
Moorman, A.V.
af5027f3-4927-4e62-b1be-ac4bdcf6cd8f
Ross, F.
ec0958f8-b992-4e4a-b7e3-c474600390ba
Mazzullo, H.
a6485fce-a86a-472b-aa8a-dbe3edab49e7
Strefford, J.C.
3782b392-f080-42bf-bdca-8aa5d6ca532f
Siebert, R.
4297324b-08e2-4ed7-8f63-43a4b18eae1b
Dyer, M.J.S.
14dd1a96-2bfb-4583-9c42-4aa54b611273
Harrison, C.J.
9c9f6b47-8bfc-49dd-b156-74539b170291

Russell, L.J., Akasaka, T., Majid, A., Sugimoto, K.J., Karran, E.L., Nagel, I., Harder, L., Claviez, A., Gesk, S., Moorman, A.V., Ross, F., Mazzullo, H., Strefford, J.C., Siebert, R., Dyer, M.J.S. and Harrison, C.J. (2008) t(6;14)(p22;q32): a new recurrent IGH@ translocation involving ID4 in B-cell precursor acute lymphoblastic leukemia (BCP-ALL). Blood, 111 (1), 387-391. (doi:10.1182/blood-2007-07-092015). (PMID:17940204)

Record type: Article

Abstract

Translocations involving the immunoglobulin heavy chain locus (IGH@) at chromosome band 14q32 are common in mature B-cell neoplasms, but are rare in B-cell precursor acute lymphoblastic leukemia (BCP-ALL). Here, we report the translocation, t(6;14)(p22;q32), involving IGH@ as a novel recurrent translocation in 13 BCP-ALL patients. Fluorescence in situ hybridization and long-distance inverse polymerase chain reaction (PCR) identified ID4 as the partner gene. Breakpoints were scattered over a 19kb region centromeric of ID4. Quantitative real-time PCR showed up-regulation of ID4 mRNA. All patients had deletions of CDKN2A and PAX5 located on the short arm of chromosome 9, frequently as a result of an isochromosome, i(9)(q10) (9/13, 69%). This study defines a new subgroup of BCP-ALL characterized by ID4 over-expression and CDKN2A and PAX5 deletions. Preliminary survival data suggest that this subgroup may be associated with a good response to therapy.

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More information

e-pub ahead of print date: 16 October 2007
Published date: 1 January 2008
Keywords: apoptosis, germany, neoplasms, human, leukemia, activation, expression, malignancies, overexpression, t(14, loop-helix proteins, survival, time, cancer, london, deletions, blood, in situ hybridization, patients, gene, report, fluorescence, england, lymphocytic-leukemia, arm, therapy, polymerase chain reaction, hematology, 19)(q32, q13), up-regulation

Identifiers

Local EPrints ID: 68133
URI: http://eprints.soton.ac.uk/id/eprint/68133
ISSN: 0006-4971
PURE UUID: 5ee211dc-a880-4f9b-b40a-bfb967732e27
ORCID for J.C. Strefford: ORCID iD orcid.org/0000-0002-0972-2881

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Date deposited: 11 Sep 2009
Last modified: 14 Mar 2024 02:49

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Contributors

Author: L.J. Russell
Author: T. Akasaka
Author: A. Majid
Author: K.J. Sugimoto
Author: E.L. Karran
Author: I. Nagel
Author: L. Harder
Author: A. Claviez
Author: S. Gesk
Author: A.V. Moorman
Author: F. Ross
Author: H. Mazzullo
Author: J.C. Strefford ORCID iD
Author: R. Siebert
Author: M.J.S. Dyer
Author: C.J. Harrison

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