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Forward transport: 14-3-3 binding overcomes retention in endoplasmic reticulum by dibasic signals

Forward transport: 14-3-3 binding overcomes retention in endoplasmic reticulum by dibasic signals
Forward transport: 14-3-3 binding overcomes retention in endoplasmic reticulum by dibasic signals
Proteins with dibasic retention motifs are subject to retrograde transport to endoplasmic reticulum (ER) by COPI-coated vesicles. As forward transport requires escape from ER retention, general release mechanisms have been expected. Here, KCNK3 potassium channels are shown to bear two cytoplasmic trafficking motifs: an N-terminal dibasic site that binds ?-COP to hold channels in ER and a C-terminal “release” site that binds the ubiquitous intracellular regulator 14-3-3? on a nonclassical motif in a phosphorylation-dependent fashion to suppress ?-COP binding and allow forward transport. The strategy appears to be common. The major histocompatibility antigen class II-associated invariant chain Iip35 exhibits dibasic retention, carries a release motif, and shows mutually exclusive binding of ?-COP and 14-3-3? on adjacent N-terminal sites. Other retained proteins are demonstrated to carry functional 14-3-3? release motifs
proteins, protein transport, research support, nerve tissue, tyrosine 3-monooxygenase, potassium channels, animals, physiology, coatomer protein, amino acid motifs, research
0092-8674
577-588
O'kelly, Ita
e640f28a-42f0-48a6-9ce2-cb5a85d08c66
Butler, Margaret
de1ff527-db81-4f3e-b57a-f37c2dd4d863
Zilberberg, Noam
6a77b1d8-b9f0-46f0-93d3-c402020039e4
Goldstein, Steve A.N.
e957cee2-71ad-4170-a086-5772415f58bc
O'kelly, Ita
e640f28a-42f0-48a6-9ce2-cb5a85d08c66
Butler, Margaret
de1ff527-db81-4f3e-b57a-f37c2dd4d863
Zilberberg, Noam
6a77b1d8-b9f0-46f0-93d3-c402020039e4
Goldstein, Steve A.N.
e957cee2-71ad-4170-a086-5772415f58bc

O'kelly, Ita, Butler, Margaret, Zilberberg, Noam and Goldstein, Steve A.N. (2002) Forward transport: 14-3-3 binding overcomes retention in endoplasmic reticulum by dibasic signals. Cell, 111 (4), 577-588. (doi:10.1016/S0092-8674(02)01040-1).

Record type: Article

Abstract

Proteins with dibasic retention motifs are subject to retrograde transport to endoplasmic reticulum (ER) by COPI-coated vesicles. As forward transport requires escape from ER retention, general release mechanisms have been expected. Here, KCNK3 potassium channels are shown to bear two cytoplasmic trafficking motifs: an N-terminal dibasic site that binds ?-COP to hold channels in ER and a C-terminal “release” site that binds the ubiquitous intracellular regulator 14-3-3? on a nonclassical motif in a phosphorylation-dependent fashion to suppress ?-COP binding and allow forward transport. The strategy appears to be common. The major histocompatibility antigen class II-associated invariant chain Iip35 exhibits dibasic retention, carries a release motif, and shows mutually exclusive binding of ?-COP and 14-3-3? on adjacent N-terminal sites. Other retained proteins are demonstrated to carry functional 14-3-3? release motifs

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More information

Published date: 15 November 2002
Keywords: proteins, protein transport, research support, nerve tissue, tyrosine 3-monooxygenase, potassium channels, animals, physiology, coatomer protein, amino acid motifs, research
Organisations: Human Genetics

Identifiers

Local EPrints ID: 71946
URI: http://eprints.soton.ac.uk/id/eprint/71946
ISSN: 0092-8674
PURE UUID: 05b09d55-6561-44a4-9c70-59c877741cde

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Date deposited: 13 Jan 2010
Last modified: 13 Mar 2024 20:52

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Contributors

Author: Ita O'kelly
Author: Margaret Butler
Author: Noam Zilberberg
Author: Steve A.N. Goldstein

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